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Role of histamine in spinal cord evoked potentials and edema following spinal cord injury: experimental observations in the rat.

作者信息

Winkler T, Sharma H S, Stålberg E, Olsson Y, Nyberg F

机构信息

Department of Clinical Neurophysiology, University Hospital, Uppsala, Sweden.

出版信息

Inflamm Res. 1995 Apr;44 Suppl 1:S44-5. doi: 10.1007/BF01674388.

DOI:10.1007/BF01674388
PMID:8520994
Abstract
摘要

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Role of histamine in spinal cord evoked potentials and edema following spinal cord injury: experimental observations in the rat.
Inflamm Res. 1995 Apr;44 Suppl 1:S44-5. doi: 10.1007/BF01674388.
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Topical application of dynorphin A (1-17) antiserum attenuates trauma induced alterations in spinal cord evoked potentials, microvascular permeability disturbances, edema formation and cell injury: an experimental study in the rat using electrophysiological and morphological approaches.强啡肽A（1-17）抗血清的局部应用可减轻创伤引起的脊髓诱发电位改变、微血管通透性紊乱、水肿形成和细胞损伤：一项在大鼠中使用电生理和形态学方法的实验研究。
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5
Carrageenan-induced oedema in the rat paw - histamine participation.角叉菜胶诱导的大鼠足爪水肿——组胺的作用
Agents Actions. 1982 Apr;12(1-2):201-2. doi: 10.1007/BF01965145.
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Indomethacin, an inhibitor of prostaglandin synthesis attenuates alteration in spinal cord evoked potentials and edema formation after trauma to the spinal cord: an experimental study in the rat.吲哚美辛，一种前列腺素合成抑制剂，可减轻脊髓损伤后脊髓诱发电位的改变和水肿形成：大鼠实验研究。
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Histamine promotes locomotion recovery after spinal cord hemisection via inhibiting astrocytic scar formation.组胺通过抑制星形胶质细胞瘢痕形成促进脊髓半切术后运动功能恢复。
CNS Neurosci Ther. 2015 May;21(5):454-62. doi: 10.1111/cns.12379. Epub 2015 Jan 24.
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Early microvascular reactions and blood-spinal cord barrier disruption are instrumental in pathophysiology of spinal cord injury and repair: novel therapeutic strategies including nanowired drug delivery to enhance neuroprotection.

本文引用的文献

1
Indomethacin, an inhibitor of prostaglandin synthesis attenuates alteration in spinal cord evoked potentials and edema formation after trauma to the spinal cord: an experimental study in the rat.吲哚美辛，一种前列腺素合成抑制剂，可减轻脊髓损伤后脊髓诱发电位的改变和水肿形成：大鼠实验研究。
Neuroscience. 1993 Feb;52(4):1057-67. doi: 10.1016/0306-4522(93)90552-q.
2
Mediators of blood-brain barrier dysfunction and formation of vasogenic brain edema.血脑屏障功能障碍和血管源性脑水肿形成的介质。
J Cereb Blood Flow Metab. 1988 Oct;8(5):621-34. doi: 10.1038/jcbfm.1988.109.
3
Role of histamine in traumatic brain edema. An experimental study in the rat.
早期微血管反应和血脊髓屏障破坏在脊髓损伤和修复的病理生理学中起着重要作用：包括纳米线药物递送在内的新的治疗策略可增强神经保护作用。
J Neural Transm (Vienna). 2011 Jan;118(1):155-76. doi: 10.1007/s00702-010-0514-4. Epub 2010 Dec 16.
组胺在创伤性脑水肿中的作用。大鼠实验研究。
J Neurol Sci. 1989 Mar;90(1):87-97. doi: 10.1016/0022-510x(89)90048-8.
4
A satellite symposium of the XIth International Congress of Pharmacology of IUPHAR. July 6-8, 1990, Noordwijkerhout, The Netherlands.国际药理学联合会（IUPHAR）第十一届国际药理学大会卫星研讨会。1990年7月6日至8日，荷兰诺德韦克豪特
Agents Actions Suppl. 1991;33:1-434.
5
Histamine modulates heat stress-induced changes in blood-brain barrier permeability, cerebral blood flow, brain oedema and serotonin levels: an experimental study in conscious young rats.组胺调节热应激诱导的血脑屏障通透性、脑血流量、脑水肿和血清素水平的变化：一项针对清醒幼鼠的实验研究。
Neuroscience. 1992 Sep;50(2):445-54. doi: 10.1016/0306-4522(92)90436-6.
6
Release of endogenous neurochemicals may increase vascular permeability, induce edema and influence cell changes in trauma to the spinal cord.
Prog Brain Res. 1992;91:197-203. doi: 10.1016/s0079-6123(08)62335-3.
7
Pharmacological strategies in CNS trauma.中枢神经系统创伤的药理学策略
Trends Pharmacol Sci. 1992 Jan;13(1):29-35. doi: 10.1016/0165-6147(92)90013-v.