al Jishi F, al Kawi M Z, el Ramahi K, Omer S
Department of Medicine (MBC 46), King Faisal Specialist Hospital and Research Centre, Riyadh, Saudi Arabia.
Lupus. 1995 Aug;4(4):321-3. doi: 10.1177/096120339500400416.
A young man with systemic lupus (SLE) developed hemichorea 13 years after the onset of his illness. For the first time in the course of his illness he had a positive test for anticardiolipin antibodies (aCL). Magnetic resonance imaging (MRI) of his brain showed lesions of presumed vascular cause in the ipsilateral basal ganglia. The findings support the contention that an immune phenomenon, invisible on proton imaging by MRI, is responsible for the striatal neuronal activation. Chorea, the clinical expression of this activation, was probably blocked on the side previously affected by vascular pathology.
一名患有系统性红斑狼疮（SLE）的年轻男性在发病13年后出现了偏侧舞蹈症。在其病程中，抗心磷脂抗体（aCL）检测首次呈阳性。他的脑部磁共振成像（MRI）显示同侧基底神经节有推测为血管源性的病变。这些发现支持了这样一种观点，即一种在MRI质子成像上不可见的免疫现象是纹状体神经元激活的原因。舞蹈症作为这种激活的临床表现，可能在先前受血管病变影响的一侧受到了抑制。
