Kuznetsov O K, Gaber V K, Lindeberg T Ia, Kostetskaia T V, Shmel'kova V I
Vopr Onkol. 1979;25(2):42-4.
Sex hormones (testosterone, estradiol) and thyroxine would activate irregularly the production of the specific group antigen (GS-antigen) of avian ribodesoxyviruses in the culture of "normal" chick embryonal cells (CEC, phenotype C/O, GS-). The mentioned hormones as well as hydrocortisone and insulin would not prevent the Rous sarcoma virus (RSV) infecting of CEC, render no essential action on the accumulation of RSV in the infected CEC culture, and failed to activate the formation of mature RSV in virogenic cultures of rat (XC, R--B77) and mice cells (DBA--B77).
性激素（睾酮、雌二醇）和甲状腺素会在“正常”鸡胚细胞（CEC，表型C/O，GS-）培养物中不规则地激活禽核糖脱氧病毒特异性群抗原（GS抗原）的产生。上述激素以及氢化可的松和胰岛素不会阻止劳斯肉瘤病毒（RSV）感染CEC，对感染的CEC培养物中RSV的积累没有实质性作用，并且未能在大鼠（XC，R--B77）和小鼠细胞（DBA--B77）的病毒发生培养物中激活成熟RSV的形成。
