Larner A J
Freeman Hospital, High Heaton, Newcastle upon Tyne.
Br J Clin Pract. 1995 Nov-Dec;49(6):331-2.
A patient whose slow-relaxing tendon reflexes returned to normal after cardiac pacing for complete heart block is reported. Since the patient was euthyroid, these observations add further weight to previous suggestions that intrinsic catecholaminergic mechanisms, as well as thyroid status, may play a role in the pathogenesis of slow-relaxing reflexes.
报告了一名完全性心脏传导阻滞患者在心脏起搏后缓慢松弛的腱反射恢复正常。由于该患者甲状腺功能正常,这些观察结果进一步支持了先前的观点,即内在的儿茶酚胺能机制以及甲状腺状态可能在缓慢松弛反射的发病机制中起作用。