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Probing the depths of degradation: matrix metalloproteinase-2 and endometrial menstrual breakdown.

作者信息

Liotta L A

出版信息

J Clin Invest. 1996 Jan 15;97(2):273-4. doi: 10.1172/JCI118412.

DOI:10.1172/JCI118412
PMID:8567944
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC507014/
Abstract
摘要

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Probing the depths of degradation: matrix metalloproteinase-2 and endometrial menstrual breakdown.探索降解的深度：基质金属蛋白酶-2与子宫内膜月经崩解
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Expert Opin Investig Drugs. 2012 Jun;21(6):797-805. doi: 10.1517/13543784.2012.681043. Epub 2012 Apr 23.
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Suppression of uPA and uPAR attenuates angiogenin mediated angiogenesis in endothelial and glioblastoma cell lines.uPA 和 uPAR 的抑制作用可减弱血管生成素介导的内皮细胞和神经胶质瘤细胞系的血管生成。
PLoS One. 2010 Aug 27;5(8):e12458. doi: 10.1371/journal.pone.0012458.
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Oxidative stress and the development of diabetic retinopathy: contributory role of matrix metalloproteinase-2.氧化应激与糖尿病视网膜病变的发展：基质金属蛋白酶-2的促成作用
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Menstrual breakdown of human endometrium can be mimicked in vitro and is selectively and reversibly blocked by inhibitors of matrix metalloproteinases.人类子宫内膜的月经崩解可在体外模拟，且会被基质金属蛋白酶抑制剂选择性地、可逆地阻断。
Proc Natl Acad Sci U S A. 1996 Aug 20;93(17):9120-5. doi: 10.1073/pnas.93.17.9120.