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VLA-4 and VLA-5 integrin expression in adult acute lymphoblastic leukemia.

作者信息

Stagno F, Cacciola E, Guglielmo P, Cacciola R R, Cacciola E

出版信息

Exp Hematol. 1996 Mar;24(4):493.

PMID:8608797
Abstract
摘要

相似文献

1
VLA-4 and VLA-5 integrin expression in adult acute lymphoblastic leukemia.
Exp Hematol. 1996 Mar;24(4):493.
2
Differential utilization of VLA-4 (alpha 4 beta 1) and -5 (alpha 5 beta 1) integrins during the development of mouse bone marrow-derived mast cells.小鼠骨髓源性肥大细胞发育过程中VLA - 4(α4β1)和VLA - 5(α5β1)整合素的差异利用
Differentiation. 1996 Sep;60(5):317-25. doi: 10.1046/j.1432-0436.1996.6050317.x.
3
VLA-5 and transendothelial migration.
Nat Med. 2002 Aug;8(8):765; author reply 765-6. doi: 10.1038/nm0802-765a.
4
Interleukin-3 and Flt3-ligand induce adhesion of Baf3/Flt3 precursor B-lymphoid cells to fibronectin via activation of VLA-4 and VLA-5.白细胞介素-3和Flt3配体通过激活VLA-4和VLA-5诱导Baf3/Flt3前体B淋巴细胞与纤连蛋白黏附。
Cell Immunol. 1998 Jul 10;187(1):27-33. doi: 10.1006/cimm.1998.1318.
5
Fibronectin increases the migration induced by stromal cell-derived factor-1 alpha (SDF-1 alpha) in pre-B acute lymphoblastic leukemia cells.纤连蛋白可增强基质细胞衍生因子-1α(SDF-1α)诱导的前B细胞急性淋巴细胞白血病细胞的迁移。
Eur Cytokine Netw. 2001 Apr-Jun;12(2):223-30.
6
Interleukin-11 enhancement of VLA-5 mediated adhesion of CD34+ cells from cord blood to fibronectin is associated with the PI-3 kinase pathway.白细胞介素-11增强脐带血中CD34+细胞通过VLA-5介导的与纤连蛋白的黏附作用,这与PI-3激酶途径相关。
In Vivo. 2000 Mar-Apr;14(2):331-7.
7
Differences in phosphatase modulation of alpha4beta1 and alpha5beta1 integrin-mediated adhesion and migration of B16F1 cells.α4β1和α5β1整合素介导的B16F1细胞黏附与迁移的磷酸酶调节差异。
Biochem Cell Biol. 1999;77(5):409-20.
8
Differential use of very late antigen-4 and -5 integrins by hematopoietic precursors and myeloma cells to adhere to transforming growth factor-beta1-treated bone marrow stroma.造血前体细胞和骨髓瘤细胞对极晚期抗原-4和-5整合素的不同利用,以黏附于经转化生长因子-β1处理的骨髓基质。
J Biol Chem. 1998 May 15;273(20):12056-60. doi: 10.1074/jbc.273.20.12056.
9
VLA-4-dependent myeloma cell adhesion.VLA - 4依赖的骨髓瘤细胞黏附。
Leuk Lymphoma. 2001 Apr;41(3-4):239-45. doi: 10.3109/10428190109057979.
10
Immature leukemic CD34+CXCR4+ cells from CML patients have lower integrin-dependent migration and adhesion in response to the chemokine SDF-1.慢性粒细胞白血病(CML)患者的未成熟白血病CD34+CXCR4+细胞对趋化因子SDF-1的反应中,整合素依赖性迁移和黏附能力较低。
Stem Cells. 2002;20(3):259-66. doi: 10.1634/stemcells.20-3-259.

引用本文的文献

1
Integrin VLA-5 and FAK are Good Targets to Improve Treatment Response in the Philadelphia Chromosome Positive Acute Lymphoblastic Leukemia.整合素 VLA-5 和 FAK 是改善费城染色体阳性急性淋巴细胞白血病治疗反应的良好靶点。
Front Oncol. 2014 May 15;4:112. doi: 10.3389/fonc.2014.00112. eCollection 2014.
2
High VLA-4 expression is associated with adverse outcome and distinct gene expression changes in childhood B-cell precursor acute lymphoblastic leukemia at first relapse.高 VLA-4 表达与儿童 B 细胞前体急性淋巴细胞白血病首次复发时的不良预后和明显的基因表达变化相关。
Haematologica. 2011 Nov;96(11):1627-35. doi: 10.3324/haematol.2011.047993. Epub 2011 Aug 9.