Wiemer J, Winkelmann B R, Beyersdorf F, Sarai K, Reifart N, Hofmann M, Klepzig H
Kardiologie/Nephrologie Klinikum J.W. Goethe-Universität, Frankfurt/Main.
Z Kardiol. 1996 Apr;85(4):221-5.
Sinus Valsalva aneurysms belong to the less common congenital or acquired structural cardiac anomalies. However, in patients with known cardiac anomalies and uncertain or uncharacteristic cardiac symptoms the existence of a sinus Valsalva aneurysm must be taken into consideration. A sinus Valsalva aneurysm can be clinically silent as in the case of the 56-year-old patient with an accompanying bacterial endocarditis. An increasing aortic regurgitation after dilatation of a coarctation of the aorta can also proceed with an ecstasy of the ascending aorta and an aneurysm of the sinus Valsalva (case 2). Furthermore, a rapid dilatation of a non-ruptured sinus Valsalva aneurysm can cause a severe compression of coronary arteries with subsequent myocardial infarction, as in the 27-year-old patient with congenital aortic stenosis and acute endocarditis in case 3.
瓦氏窦瘤属于较罕见的先天性或后天性心脏结构异常。然而，对于已知存在心脏异常且有不确定或不典型心脏症状的患者，必须考虑瓦氏窦瘤的存在。瓦氏窦瘤在临床上可能没有症状，比如一名56岁伴有细菌性心内膜炎的患者。主动脉缩窄扩张后主动脉反流增加也可能伴有升主动脉扩张和瓦氏窦瘤（病例2）。此外，未破裂的瓦氏窦瘤快速扩张可导致冠状动脉严重受压，继而引发心肌梗死，如病例3中一名患有先天性主动脉瓣狭窄和急性心内膜炎的27岁患者。
