Brogren E, Hadders-Algra M, Forssberg H
Department of Woman and Child Health, Karolinska Institute, Stockholm, Sweden.
Dev Med Child Neurol. 1996 May;38(5):379-88. doi: 10.1111/j.1469-8749.1996.tb15095.x.
To clarify the neural mechanisms controlling equilibrium during sitting, and the implications for the optimal sitting position for children with CP, automatic postural adjustments after perturbations of the support surface during sitting were investigated in seven children with spastic diplegia and in seven age-matched controls. A sudden backward sway of the body evoked brisk responses in ¿ventral' muscles in both groups. However, the order of muscle recruitment, which in most non-disabled children was caudal, was reversed in the children with diplegia, whose first response was in the neck flexors. The children with diplegia also co-activated antagonistic neck and hip muscles. The authors concluded that although children with spastic diplegia may produce a basic muscle activation pattern (first level of the central pattern generator, CPG), they cannot adjust the pattern in response to external changes (second level of the CPG).
为了阐明控制坐姿平衡的神经机制以及对痉挛性双瘫儿童最佳坐姿的影响，对7名痉挛性双瘫儿童和7名年龄匹配的对照组儿童在坐姿时支撑面受到扰动后的自动姿势调整进行了研究。两组中，身体突然向后摇晃均引起“腹侧”肌肉的快速反应。然而，在大多数非残疾儿童中按尾侧顺序的肌肉募集顺序，在双瘫儿童中却相反，他们的第一反应是颈部屈肌。双瘫儿童还共同激活了拮抗的颈部和髋部肌肉。作者得出结论，尽管痉挛性双瘫儿童可能产生基本的肌肉激活模式（中枢模式发生器的第一级，CPG），但他们无法根据外部变化调整该模式（CPG的第二级）。
