动脉粥样硬化的免疫学：小鼠模型的前景 - Suppr

Suppr

超能文献

Immunology of atherosclerosis: the promise of mouse models.

作者信息

Lichtman A H, Cybulsky M, Luscinskas F W

机构信息

Department of Pathology, Brigham and Women's Hospital, Boston, Massachusetts 02115, USA.

出版信息

Am J Pathol. 1996 Aug;149(2):351-7.

PMID:8701975

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1865310/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/5a43/1865310/66ee275c3b87/amjpathol00032-0010-a.jpg

相似文献

Immunology of atherosclerosis: the promise of mouse models.动脉粥样硬化的免疫学：小鼠模型的前景

Am J Pathol. 1996 Aug;149(2):351-7.

Are immunological mechanisms relevant for the development of atherosclerosis?免疫机制与动脉粥样硬化的发展是否相关？

Clin Immunol. 1999 Feb;90(2):153-6. doi: 10.1006/clim.1998.4682.

[Cytokines in the pathogenesis of atherosclerosis].[细胞因子在动脉粥样硬化发病机制中的作用]

Dtsch Med Wochenschr. 2002 Jan 18;127(3):94-9. doi: 10.1055/s-2002-19597.

Viruses and atherosclerosis.病毒与动脉粥样硬化

Isr J Med Sci. 1992 Jul;28(7):463-5.

Animal models of atherosclerosis.

Adv Vet Sci Comp Med. 1972;16:151-73.

Atherosclerosis as an immune disease?动脉粥样硬化是一种免疫疾病？

Med Hypotheses. 1991 Sep;36(1):24-6. doi: 10.1016/0306-9877(91)90159-v.

[The role of immunological factors in the pathogenesis of arteriosclerosis].

Postepy Hig Med Dosw. 1991;45(3-4):187-98.

[Contemporary views on the pathogenesis of atherosclerosis].[关于动脉粥样硬化发病机制的当代观点]

Vestn Ross Akad Med Nauk. 2006(9-10):66-74.

Immunization of low-density lipoprotein receptor deficient (LDL-RD) mice with heat shock protein 65 (HSP-65) promotes early atherosclerosis.用热休克蛋白65（HSP - 65）对低密度脂蛋白受体缺陷（LDL - RD）小鼠进行免疫会促进早期动脉粥样硬化。

J Autoimmun. 2000 Mar;14(2):115-21. doi: 10.1006/jaut.1999.0351.

The contribution of avian models to our understanding of atherosclerosis and their promise for the future.禽类模型对我们理解动脉粥样硬化的贡献及其未来前景。

Lab Anim Sci. 1998 Dec;48(6):565-8.

引用本文的文献

Adaptive immunity and atherosclerosis: mouse tales in the AJP.适应性免疫与动脉粥样硬化：《美国生理学杂志》中的小鼠故事。

Am J Pathol. 2013 Jan;182(1):5-9. doi: 10.1016/j.ajpath.2012.10.006. Epub 2012 Nov 16.

G2A deficiency in mice promotes macrophage activation and atherosclerosis.小鼠中的G2A缺陷会促进巨噬细胞活化和动脉粥样硬化。

Circ Res. 2009 Feb 13;104(3):318-27. doi: 10.1161/CIRCRESAHA.108.181131. Epub 2008 Dec 23.

Chlamydia pneumoniae exacerbates aortic inflammatory foci caused by murine cytomegalovirus infection in normocholesterolemic mice.肺炎衣原体可加剧正常胆固醇水平小鼠中由鼠巨细胞病毒感染引起的主动脉炎症病灶。

Clin Diagn Lab Immunol. 2001 Nov;8(6):1263-6. doi: 10.1128/CDLI.8.6.1263-1266.2001.

Lymphocytes are important in early atherosclerosis.淋巴细胞在早期动脉粥样硬化中起重要作用。

J Clin Invest. 2001 Jul;108(2):251-9. doi: 10.1172/JCI11380.

The effects of total lymphocyte deficiency on the extent of atherosclerosis in apolipoprotein E-/- mice.全淋巴细胞缺乏对载脂蛋白E基因敲除小鼠动脉粥样硬化程度的影响。

J Clin Invest. 1997 Sep 15;100(6):1575-80. doi: 10.1172/JCI119681.

本文引用的文献

Changes in the endothelium of the aorta and the behaviour of macrophages in experimental atheroma of rabbits.兔实验性动脉粥样硬化中主动脉内皮的变化及巨噬细胞的行为

J Pathol Bacteriol. 1958 Apr;75(2):245-51. doi: 10.1002/path.1700750202.

Evidence for a local immune response in atherosclerosis. CD4+ T cells infiltrate lesions of apolipoprotein-E-deficient mice.动脉粥样硬化中局部免疫反应的证据。CD4 + T细胞浸润载脂蛋白E缺乏小鼠的病变部位。

Am J Pathol. 1996 Aug;149(2):359-66.

Antigen-presenting cells. Professionals and amateurs.抗原呈递细胞。专业者与非专业者。

Curr Biol. 1995 Oct 1;5(10):1095-7. doi: 10.1016/s0960-9822(95)00219-3.

Apolipoprotein E and the apolipoprotein E-deficient mouse.

Annu Rev Nutr. 1995;15:495-518. doi: 10.1146/annurev.nu.15.070195.002431.

Increased expression of heat shock protein 65 coincides with a population of infiltrating T lymphocytes in atherosclerotic lesions of rabbits specifically responding to heat shock protein 65.热休克蛋白65表达增加与兔动脉粥样硬化病变中一群对热休克蛋白65有特异性反应的浸润性T淋巴细胞同时出现。

J Clin Invest. 1993 Jun;91(6):2693-702. doi: 10.1172/JCI116508.

Accelerated atherosclerosis in hyperlipidemic C57BL/6 mice treated with cyclosporin A.用环孢素A治疗的高脂血症C57BL/6小鼠的动脉粥样硬化加速。

Am J Pathol. 1993 Jun;142(6):1906-15.

The pathogenesis of atherosclerosis: a perspective for the 1990s.动脉粥样硬化的发病机制：20世纪90年代的展望

Nature. 1993 Apr 29;362(6423):801-9. doi: 10.1038/362801a0.

Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery.低密度脂蛋白受体基因敲除小鼠的高胆固醇血症及其通过腺病毒介导的基因递送的逆转。

J Clin Invest. 1993 Aug;92(2):883-93. doi: 10.1172/JCI116663.

Atherosclerosis in transgenic mice overexpressing apolipoprotein A-II.过表达载脂蛋白A-II的转基因小鼠中的动脉粥样硬化

Science. 1993 Jul 23;261(5120):469-72. doi: 10.1126/science.8332912.

Atherosclerosis in mice lacking apo E. Evaluation of lesional development and progression.缺乏载脂蛋白E的小鼠的动脉粥样硬化。病变发展和进展的评估。

Arterioscler Thromb. 1994 Jan;14(1):141-7. doi: 10.1161/01.atv.14.1.141.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验