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类风湿关节炎中寰枢椎稳定性半脱位的发病机制与患病率

Pathogenetic mechanism and prevalence of the stable atlantoaxial subluxation in rheumatoid arthritis.

作者信息

Kauppi M, Sakaguchi M, Konttinen Y T, Hakala M

机构信息

Rheumatism Foundation Hospital, Heinola, Finland.

出版信息

J Rheumatol. 1996 May;23(5):831-4.

PMID:8724293
Abstract

OBJECTIVE

To study the pathogenetic mechanisms responsible for and prevalence of stable atlantoaxial subluxation (AAS).

METHODS

Lateral view cervical spine radiographs during flexion and extension were studied in 262 patients with rheumatoid arthritis (RA).

RESULTS

AAS was found in 130 patients, 42 (32%) of whom had a stable deformity; in a population based subseries, stable AAS was seen in 16% of the patients with ASS. Atlantoaxial impaction (AAI) was diagnosed in 98 cases and in 37 (88%) of the 42 patients with stable AAS.

CONCLUSION

The main cause of the spontaneous stabilization and occasional ankylosis of the atlantoaxial area is the development of erosions and collapse in the lateral facet joints leading to AAI.

摘要

目的

研究稳定型寰枢椎半脱位(AAS)的发病机制及患病率。

方法

对262例类风湿关节炎(RA)患者的颈椎屈伸位侧位X线片进行研究。

结果

130例患者发现有AAS,其中42例(32%)存在稳定畸形;在一个基于人群的亚组系列中,16%的寰枢椎半脱位(ASS)患者出现稳定型AAS。98例诊断为寰枢椎撞击症(AAI),42例稳定型AAS患者中有37例(88%)诊断为AAI。

结论

寰枢椎区域自发稳定和偶尔发生强直的主要原因是外侧小关节出现侵蚀和塌陷,导致AAI。

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