Reis S M, Magalhaes L A, de Carvalho J F
Bolsista da FAPESP-Campinas, SP-Brasil.
Rev Saude Publica. 1995 Aug;29(4):259-64. doi: 10.1590/s0034-89101995000400001.
The resistance of B. tenagophila snails to infection by Schistosoma mansoni was studied. These snails had been previously inoculated with hemolymph talsen from other snails infected by S. mansoni of SJ strain or by other trematodes. The findings suggest that two types--cellular and humoral--are responsible for the resistance to infection by S. mansoni. The cellular response occurred when the snails were inoculated with hemolymph from snails infected by S. mansoni, B. tenagophila inoculate with hemolymph from snails infected by furcocercariae without eye-spot, also showed resistance to the infection by S. mansoni. In this latter case, the degenerated sporocysts were not enveloped by amebocitary reaction, leading to the conclusion that humoral factors could be involved.
对嗜眼吸虫蜗牛感染曼氏血吸虫的抗性进行了研究。这些蜗牛之前已接种过来自感染了SJ株曼氏血吸虫或其他吸虫的其他蜗牛的血淋巴。研究结果表明，细胞和体液两种类型对抵抗曼氏血吸虫感染起作用。当蜗牛接种来自感染曼氏血吸虫的蜗牛的血淋巴时会发生细胞反应，接种来自感染无眼点尾蚴的蜗牛的血淋巴的嗜眼吸虫蜗牛，对曼氏血吸虫感染也表现出抗性。在后一种情况下，退化的包蚴未被阿米巴细胞反应包裹，从而得出体液因子可能参与其中的结论。
