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双侧后皮质萎缩或梗死引起的巴林特综合征:康复策略及其局限性

Balint's syndrome arising from bilateral posterior cortical atrophy or infarction: rehabilitation strategies and their limitation.

作者信息

Perez F M, Tunkel R S, Lachmann E A, Nagler W

机构信息

Department of Physical Medicine and Rehabilitation, Staten Island University Hospital North, New York 10305, USA.

出版信息

Disabil Rehabil. 1996 Jun;18(6):300-4. doi: 10.3109/09638289609165884.

Abstract

Balint's syndrome is characterized by faulty visual scanning, dysmetria secondary to a visual perceptual deficit, and an inability to recognize more than one object at a time. We report three cases of Balint's syndrome and the individualized rehabilitation they received. One patient developed symptoms of Balint's syndrome caused by bilateral occipitoparietal infarcts. In the two other cases the symptoms arose secondary to posterior cortical atrophy, a slowly progressive dementia with alexia and agraphia. All three patients initially responded to a multicontext treatment approach with intensive verbal cueing and organizational strategies with subsequent improvement in visual recognition, reaching and scanning. Continued deterioration from posterior cortical atrophy or a second infarction resulted in worsening dementia in two patients. Cognitive remediation was required as visual perceptual rehabilitation became ineffective. Clinicians should be familiar with Balint's syndrome and its various aetiologies. Lack of awareness of this syndrome may lead to a misdiagnosis and resulting inappropriate or inadequate treatment.

摘要

巴林特综合征的特征为视觉扫描障碍、继发于视觉感知缺陷的辨距不良以及一次无法识别多个物体。我们报告三例巴林特综合征病例及其接受的个体化康复治疗。一名患者因双侧枕顶叶梗死出现巴林特综合征症状。在另外两例中,症状继发于后部皮质萎缩,这是一种伴有失读症和失写症的缓慢进展性痴呆。所有三名患者最初对多情境治疗方法有反应,采用强化言语提示和组织策略,随后视觉识别、伸手和扫描能力有所改善。两名患者因后部皮质萎缩或再次梗死导致病情持续恶化,痴呆加重。由于视觉感知康复无效,需要进行认知矫正。临床医生应熟悉巴林特综合征及其各种病因。对该综合征缺乏认识可能导致误诊,进而导致不适当或不充分的治疗。

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