Hiejima K, Satake S, Sakamoto Y, Suzuki F, Sano T
Chest. 1977 Aug;72(2):170-5. doi: 10.1378/chest.72.2.170.
Two patients with a history of paroxysmal supraventricular tachycardia but no evidence of the Wolff-Parkinson-White syndrome in the conventional electrocardiograms were studied utilizing atrial and ventricular extrastimuli. The evidence obtained in these patients was consistent with a concealed Wolff-Parkinson-White syndrome. In one patient, ventricular extrastimuli revealed three different responses in the curve of ventriculoatrial conduction, depending upon the interval of stimulus. In the other patient, intracardiac recordings, including the left atrial potential, showed that paroxysmal supraventricular tachycardia was induced by an impulse via an anomalous bypass which diverged from the main bypass or stemmed independently from the left ventricle and was maintained by impulses via the main bypass which lay between the left ventricle and the left atrium. The mechanisms for initiation of the paroxysmal supraventricular tachycardia were discussed.
对两名有阵发性室上性心动过速病史但常规心电图无预激综合征证据的患者,利用心房和心室期外刺激进行了研究。这些患者获得的证据与隐匿性预激综合征相符。在一名患者中,心室期外刺激在室房传导曲线中显示出三种不同的反应,这取决于刺激间期。在另一名患者中,包括左心房电位在内的心内记录显示,阵发性室上性心动过速是由一个冲动通过一条异常旁路诱发的,该旁路从主要旁路分出或独立起源于左心室,并由通过位于左心室和左心房之间的主要旁路的冲动维持。讨论了阵发性室上性心动过速的起始机制。