Tuma R F, Lindbom L, Arfors K E
Am J Physiol. 1977 Aug;233(2):H289-94. doi: 10.1152/ajpheart.1977.233.2.H289.
Red blood cell velocity was measured in capillaries of the rabbit tenuissimus muscle during exposure to a low-oxygen-tension (PO2 = 5 mmHg) and a high-oxygen-tension (room-air PO2 = 150 mmHg) suffusion solution. Control capillary red blood cell velocity was significantly reduced (44%) by elevating the suffusion solution PO2 from 5 to 150 mmHg. The reactive hyperemias that occurred after a 120-s aortic occlusion under these two conditions were compared. The mean RBC velocity during the hyperemia in the 1st min following the removal of occlusion was significantly reduced by increasing oxygen tension, as was the duration of the hyperemia. Peak capillary red blood cell velocities in the hyperemic phase during exposures to low and high PO2 were not significantly different. It can be concluded from this study that although oxygen tension does affect postocclusive reactive hyperemia, other factors such as myogenically induced vascular relaxation also contribute to the production of this phenomenon.
在将低氧张力（PO2 = 5 mmHg）和高氧张力（室内空气PO2 = 150 mmHg）的灌注溶液注入家兔薄肌毛细血管期间，对红细胞速度进行了测量。将灌注溶液的PO2从5 mmHg提高到150 mmHg时，对照毛细血管红细胞速度显著降低（44%）。比较了在这两种条件下120秒主动脉闭塞后出现的反应性充血情况。去除闭塞后第1分钟充血期间的平均红细胞速度因氧张力增加而显著降低，充血持续时间也是如此。低PO2和高PO2暴露期间充血期的毛细血管红细胞峰值速度无显著差异。从这项研究可以得出结论，尽管氧张力确实会影响闭塞后反应性充血，但其他因素，如肌源性诱导的血管舒张，也有助于这种现象的产生。
