Taniguchi Y, Yoshida M, Tamaki T
Department of Orthopaedic Surgery, Wakayama Medical College, Japan.
J Hand Surg Am. 1996 Sep;21(5):870-4. doi: 10.1016/S0363-5023(96)80206-1.
Nine patients (11 elbows) presented with cubital tunnel syndrome that was complicated by calcium pyrophosphate dihydrate (CPPD) crystal deposition disease. Their age ranged from 63 to 86 years, and 8 of the 9 patients were more than 70 years old. X-ray film examination demonstrated the presence of CPPD crystal deposition not only in the elbow but also in the knee, wrist, or spine in all patients. Medial epicondylectomy was performed in 8 patients (9 elbows), while 1 patient with both elbows affected was treated conservatively at his request. Thickening of the ulnar collateral ligament and synovium due to CPPD crystal deposition and degenerative joint changes induced protrusion of the floor of the cubital tunnel, resulting in ulnar neuropathy in these patients. The ulnar nerve was compressed between the protruded floor and fibrous band. Though various causes of cubital tunnel syndrome have been reported, findings of this study indicate that CPPD crystal deposition disease should also be considered a cause of this syndrome.
9例患者（11个肘关节）表现为伴有二水焦磷酸钙（CPPD）晶体沉积病的肘管综合征。他们的年龄在63岁至86岁之间，9例患者中有8例年龄超过70岁。X线片检查显示，所有患者不仅在肘部，而且在膝部、腕部或脊柱均存在CPPD晶体沉积。8例患者（9个肘关节）接受了内侧上髁切除术，而1例双侧肘关节受累的患者因个人要求接受了保守治疗。CPPD晶体沉积和退行性关节改变导致尺侧副韧带和滑膜增厚，引起肘管底部突出，导致这些患者出现尺神经病变。尺神经被突出的底部和纤维带压迫。尽管已有报道肘管综合征有多种病因，但本研究结果表明，CPPD晶体沉积病也应被视为该综合征的一个病因。
