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环孢素A对肾上皮细胞系NBL-1中钠钾ATP酶表达的影响。

Effects of cyclosporine A on Na,K-ATPase expression in the renal epithelial cell line NBL-1.

作者信息

Ferrer-Martínez A, Felipe A, Barceló P, Casado F J, Ballarín J, Pastor-Anglada M

机构信息

Departament de Bioquímica i Biologia Molecular, Universitat de Barcelona, Spain.

出版信息

Kidney Int. 1996 Nov;50(5):1483-9. doi: 10.1038/ki.1996.462.

DOI:10.1038/ki.1996.462
PMID:8914013
Abstract

The bovine renal epithelial cell line NBL-1 has been used to monitor the effects of cyclosporine A (CsA) on Na+,K(+)-ATPase activity and expression. CsA at two single doses (0.6 mg/liter and 2.5 mg/liter) inhibits the ouabain-sensitive component of Rb+ uptake, assumed to be Na+,K(+)-ATPase, but increases the low activity of a furosemide-sensitive component corresponding to a Na+/K+/Cl- cotransporter. CsA addition also induces a slight decrease of alpha 1 subunit mRNA levels, without altering the already low beta 1 subunit mRNA amounts. Hypertonic treatment of NBL-1 cells leads to a significant increase in both Na+,K(+)-ATPase activity and alpha 1 subunit mRNA amounts, but does not modify beta 1 subunit mRNA levels. The differential response of the alpha 1 and beta 1 subunit genes may explain why hypertonic treatment does not result in higher alpha 1 protein expression, and supports the view that increased activity relies upon post-translational events, despite the likely transcriptional activation of the alpha 1 subunit gene. The addition of CsA does not alter the hypertonicity-mediated increase of Na+,K(+)-ATPase activity but blocks the accumulation of alpha 1 subunit mRNA. In conclusion, CsA may compromise the ion handling by renal cells as a result of the inhibition of basal Na+,K(+)-ATPase activity and the stimulation of Na+/K+/Cl- cotransport activity. Moreover, this is the first report showing that CsA may affect the long-term adaptation of the pump by altering its subunit gene expression.

摘要

牛肾上皮细胞系NBL-1已被用于监测环孢素A(CsA)对Na +,K(+)-ATP酶活性和表达的影响。两种单剂量(0.6毫克/升和2.5毫克/升)的CsA抑制了铷摄取中对哇巴因敏感的成分(假定为Na +,K(+)-ATP酶),但增加了对应于Na + / K + / Cl - 协同转运蛋白的速尿敏感成分的低活性。添加CsA还会导致α1亚基mRNA水平略有下降,而不会改变本就较低的β1亚基mRNA量。对NBL-1细胞进行高渗处理会导致Na +,K(+)-ATP酶活性和α1亚基mRNA量均显著增加,但不会改变β1亚基mRNA水平。α1和β1亚基基因的不同反应可能解释了为什么高渗处理不会导致更高的α1蛋白表达,并支持这样一种观点,即尽管α1亚基基因可能发生转录激活,但活性增加依赖于翻译后事件。添加CsA不会改变高渗介导的Na +,K(+)-ATP酶活性增加,但会阻止α1亚基mRNA的积累。总之,由于抑制基础Na +,K(+)-ATP酶活性和刺激Na + / K + / Cl - 协同转运活性,CsA可能会损害肾细胞的离子处理能力。此外,这是第一份表明CsA可能通过改变其亚基基因表达来影响泵的长期适应性的报告。

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