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Th cell development and regulation in experimental cutaneous leishmaniasis.

作者信息

Scott P

机构信息

Department of Pathobiology, University of Pennsylvania, Philadelphia, USA.

出版信息

Chem Immunol. 1996;63:98-114.

PMID:8934833
Abstract
摘要

相似文献

1
Th cell development and regulation in experimental cutaneous leishmaniasis.实验性皮肤利什曼病中辅助性T细胞的发育与调控
Chem Immunol. 1996;63:98-114.
2
Notch1 expression on T cells is not required for CD4+ T helper differentiation.T细胞上的Notch1表达对于CD4+辅助性T细胞分化并非必需。
Eur J Immunol. 2004 Jun;34(6):1588-96. doi: 10.1002/eji.200324337.
3
SOCS5 is expressed in primary B and T lymphoid cells but is dispensable for lymphocyte production and function.SOCS5在原发性B淋巴细胞和T淋巴细胞中表达，但对于淋巴细胞的产生和功能并非必需。
Mol Cell Biol. 2004 Jul;24(13):6094-103. doi: 10.1128/MCB.24.13.6094-6103.2004.
4
The mechanism of in vitro T helper cell type 1 to T helper cell type 2 switching in highly polarized Leishmania major-specific T cell populations.高度极化的利什曼原虫主要特异性T细胞群体中体外1型辅助性T细胞向2型辅助性T细胞转换的机制。
J Immunol. 1997 Feb 15;158(4):1559-64.
5
Experimental cutaneous Leishmaniasis: a powerful model to study in vivo the mechanisms underlying genetic differences in Th subset differentiation.实验性皮肤利什曼病：一种用于在体内研究Th亚群分化遗传差异潜在机制的强大模型。
Eur J Dermatol. 2002 Jul-Aug;12(4):316-8.
6
Genetic control of T helper subset differentiation in Leishmania major infection of mice.小鼠利什曼原虫主要感染中辅助性T细胞亚群分化的遗传控制
Microbes Infect. 1999 Jan;1(1):89-94. doi: 10.1016/s1286-4579(99)80019-7.
7
Characterization of T cell-mediated responses in nonhealing and healing Leishmania major infections in the absence of endogenous IL-4.内源性白细胞介素-4缺乏时，非愈合性和愈合性硕大利什曼原虫感染中T细胞介导反应的特征分析
J Immunol. 1997 Oct 1;159(7):3434-43.
8
Resistance to Leishmania major in mice.小鼠对硕大利什曼原虫的抗性。
Science. 1996 Nov 22;274(5291):1392-3.
9
Switch of CD4+ T cell differentiation from Th2 to Th1 by treatment with cathepsin B inhibitor in experimental leishmaniasis.在实验性利什曼病中，通过组织蛋白酶B抑制剂治疗使CD4 + T细胞分化从Th2向Th1转变。
J Immunol. 1998 Sep 1;161(5):2120-7.
10
T helper differentiation in resistant and susceptible B7-deficient mice infected with Leishmania major.
Eur J Immunol. 2002 Jun;32(6):1764-72. doi: 10.1002/1521-4141(200206)32:6<1764::AID-IMMU1764>3.0.CO;2-V.

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Persistent parasites and immunologic memory in cutaneous leishmaniasis: implications for vaccine designs and vaccination strategies.皮肤利什曼病中的持续性寄生虫与免疫记忆：对疫苗设计和接种策略的启示
Immunol Res. 2008;41(2):123-36. doi: 10.1007/s12026-008-8016-2.
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Development and regulation of cell-mediated immunity in experimental leishmaniasis.实验性利什曼病中细胞介导免疫的发育与调节
Immunol Res. 2003;27(2-3):489-98. doi: 10.1385/IR:27:2-3:489.
6
Gender is a major determinant of the clinical evolution and immune response in hamsters infected with Leishmania spp.性别是感染利什曼原虫属的仓鼠临床病程和免疫反应的主要决定因素。
Infect Immun. 2002 May;70(5):2288-96. doi: 10.1128/IAI.70.5.2288-2296.2002.
7
Influence of parasite load on the ability of type 1 T cells to control Leishmania major infection.寄生虫负荷对1型T细胞控制硕大利什曼原虫感染能力的影响。
Infect Immun. 2002 Feb;70(2):498-503. doi: 10.1128/IAI.70.2.498-503.2002.
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IL-4: role in disease and regulation of production.白细胞介素-4：在疾病中的作用及产生的调节
Clin Exp Immunol. 1998 Sep;113(3):317-9. doi: 10.1046/j.1365-2249.1998.00690.x.
9
Differential regulation of the interleukin-12 receptor during the innate immune response to Leishmania major.利什曼原虫主要种天然免疫反应过程中白细胞介素-12受体的差异调节
Infect Immun. 1998 Aug;66(8):3818-24. doi: 10.1128/IAI.66.8.3818-3824.1998.
10
Differentiation, regulation, and death of T helper cell subsets during infection with Leishmania major.利什曼原虫主要感染期间辅助性T细胞亚群的分化、调控及死亡
Immunol Res. 1998;17(1-2):229-38. doi: 10.1007/BF02786447.