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人类免疫缺陷病毒1型巨噬细胞嗜性和淋巴细胞中合胞体形成的独立V3包膜决定簇定位

Mapping of independent V3 envelope determinants of human immunodeficiency virus type 1 macrophage tropism and syncytium formation in lymphocytes.

作者信息

Chesebro B, Wehrly K, Nishio J, Perryman S

机构信息

Laboratory of Persistent Viral Diseases, Rocky Mountain Laboratories, National Institute of Allergy and Infectious Diseases, Hamilton, Montana 59840, USA.

出版信息

J Virol. 1996 Dec;70(12):9055-9. doi: 10.1128/JVI.70.12.9055-9059.1996.

Abstract

The V3 region of the human immunodeficiency virus type 1 (HIV-1) envelope protein is known to have a major influence on macrophage tropism as well as the ability to cause syncytium formation or fusion in CD4-positive lymphocyte cultures. Using infectious molecular HIV-1 clones, a series of mutant clones was created which allowed detailed mapping of V3 amino acid positions involved in these properties. In these experiments the non-syncytium-inducing phenotype in T cells did not always correlate with macrophage tropism. Macrophage tropism appeared to depend on the presence of certain combinations of amino acids at five specific positions within and just outside of the V3 loop itself, whereas syncytium formation in lymphocytes was influenced by substitution of particular residues at two to four positions within V3. In most cases, different V3 amino acid positions were found to independently influence macrophage tropism and syncytium formation in T cells and position 13 was the only V3 location which appeared to simultaneously influence both macrophage tropism and syncytium formation in lymphocytes.

摘要

已知人类免疫缺陷病毒1型(HIV-1)包膜蛋白的V3区对巨噬细胞嗜性以及在CD4阳性淋巴细胞培养物中引起合胞体形成或融合的能力有重大影响。利用具有感染性的分子HIV-1克隆,构建了一系列突变克隆,从而能够详细定位与这些特性相关的V3氨基酸位置。在这些实验中,T细胞中的非合胞体诱导表型并不总是与巨噬细胞嗜性相关。巨噬细胞嗜性似乎取决于V3环本身及其外侧五个特定位置上某些氨基酸组合的存在,而淋巴细胞中的合胞体形成则受V3内两到四个位置上特定残基取代的影响。在大多数情况下,发现不同的V3氨基酸位置独立影响T细胞中的巨噬细胞嗜性和合胞体形成,而位置13是唯一似乎同时影响淋巴细胞中巨噬细胞嗜性和合胞体形成的V3位置。

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