缺血性心脏中的葡萄糖代谢。 - Suppr

Suppr

超能文献

Glucose metabolism in the ischemic heart.

作者信息

Lopaschuk G D, Stanley W C

出版信息

Circulation. 1997 Jan 21;95(2):313-5. doi: 10.1161/01.cir.95.2.313.

DOI:10.1161/01.cir.95.2.313

PMID:9008441

Abstract

摘要

相似文献

Glucose metabolism in the ischemic heart.缺血性心脏中的葡萄糖代谢。

Circulation. 1997 Jan 21;95(2):313-5. doi: 10.1161/01.cir.95.2.313.

Cellular and molecular regulation of cardiac glucose transport.心脏葡萄糖转运的细胞与分子调节

J Nucl Cardiol. 2000 May-Jun;7(3):267-76. doi: 10.1016/s1071-3581(00)70016-x.

Regulation of myocardial glucose uptake and transport during ischemia and energetic stress.缺血和能量应激期间心肌葡萄糖摄取与转运的调节

Am J Cardiol. 1999 Jun 17;83(12A):25H-30H. doi: 10.1016/s0002-9149(99)00253-2.

Ischemia induces translocation of the insulin-responsive glucose transporter GLUT4 to the plasma membrane of cardiac myocytes.局部缺血会促使胰岛素反应性葡萄糖转运蛋白GLUT4易位至心肌细胞的质膜。

Circulation. 1994 Feb;89(2):793-8. doi: 10.1161/01.cir.89.2.793.

Increased sarcolemmal glucose transporter abundance in myocardial ischemia.心肌缺血时肌膜葡萄糖转运体丰度增加。

Am J Cardiol. 1997 Aug 4;80(3A):77A-84A. doi: 10.1016/s0002-9149(97)00460-8.

Myocardial glucose transporter GLUT1: translocation induced by insulin and ischemia.心肌葡萄糖转运蛋白GLUT1：由胰岛素和缺血诱导的易位

J Mol Cell Cardiol. 1999 Jul;31(7):1337-44. doi: 10.1006/jmcc.1999.0965.

Responses of GLUT4-deficient hearts to ischemia underscore the importance of glycolysis.葡萄糖转运蛋白4缺乏的心脏对缺血的反应突出了糖酵解的重要性。

Circulation. 2001 Jun 19;103(24):2961-6. doi: 10.1161/01.cir.103.24.2961.

Low-flow ischemia leads to translocation of canine heart GLUT-4 and GLUT-1 glucose transporters to the sarcolemma in vivo.低流量缺血导致犬心脏葡萄糖转运蛋白4（GLUT-4）和葡萄糖转运蛋白1（GLUT-1）在体内向肌膜移位。

Circulation. 1997 Jan 21;95(2):415-22. doi: 10.1161/01.cir.95.2.415.

Persistent myocardial ischemia increases GLUT1 glucose transporter expression in both ischemic and non-ischemic heart regions.

J Mol Cell Cardiol. 1997 Jun;29(6):1675-85. doi: 10.1006/jmcc.1997.0405.

Action of metformin on glucose transport and glucose transporter GLUT1 and GLUT4 in heart muscle cells from healthy and diabetic rats.二甲双胍对健康和糖尿病大鼠心肌细胞中葡萄糖转运及葡萄糖转运蛋白GLUT1和GLUT4的作用。

Endocrinology. 1995 Feb;136(2):412-20. doi: 10.1210/endo.136.2.7835271.

引用本文的文献

IGFBP5 mediates the therapeutic effect of isoliquiritigenin in myocardial ischemia-reperfusion injury via AKT/GLUT4 regulated insulin resistance.胰岛素样生长因子结合蛋白5通过AKT/GLUT4调节的胰岛素抵抗介导异甘草素对心肌缺血再灌注损伤的治疗作用。

Front Pharmacol. 2025 Apr 29;16:1544869. doi: 10.3389/fphar.2025.1544869. eCollection 2025.

Cardioprotective strategies in myocardial ischemia-reperfusion injury: Implications for improving clinical translation.心肌缺血再灌注损伤中的心脏保护策略：对改善临床转化的意义。

J Mol Cell Cardiol Plus. 2024 Dec 16;11:100278. doi: 10.1016/j.jmccpl.2024.100278. eCollection 2025 Mar.

Cardiac ischaemia/reperfusion in pigs and mice increases cardiomyocyte Krüppel-like factor 5 that aggravates tissue injury and remodelling.猪和小鼠的心脏缺血/再灌注会增加心肌细胞中的Krüppel样因子5，从而加重组织损伤和重塑。

Cardiovasc Res. 2025 Jun 12;121(6):900-914. doi: 10.1093/cvr/cvaf040.

Loss of PKM2 dysregulates inflammatory signaling in the infarcted murine heart.丙酮酸激酶M2（PKM2）的缺失会使梗死小鼠心脏中的炎症信号传导失调。

Physiol Rep. 2025 Jan;13(1):e70193. doi: 10.14814/phy2.70193.

Elevated Glucose on Admission Was an Independent Risk Factor for 30-Day Major Adverse Cardiovascular Events in Patients with STEMI but Not NSTEMI.入院时血糖升高是ST段抬高型心肌梗死患者30天主要不良心血管事件的独立危险因素，但非ST段抬高型心肌梗死患者并非如此。

Rev Cardiovasc Med. 2024 Jan 29;25(2):46. doi: 10.31083/j.rcm2502046. eCollection 2024 Feb.

A Feasibility Study of [F]F-AraG Positron Emission Tomography (PET) for Cardiac Imaging-Myocardial Viability in Ischemia-Reperfusion Injury Model.[F]F-AraG正电子发射断层扫描（PET）用于心脏成像——缺血再灌注损伤模型中心肌存活情况的可行性研究

Mol Imaging Biol. 2024 Oct;26(5):869-878. doi: 10.1007/s11307-024-01932-y. Epub 2024 Jul 26.

A feasibility study of [18F]F-AraG positron emission tomography (PET) for cardiac imaging - myocardial viability in ischemia-reperfusion injury model.[18F]F-阿糖鸟苷正电子发射断层扫描（PET）用于心脏成像——缺血再灌注损伤模型中心肌存活情况的可行性研究。

Res Sq. 2024 Apr 30:rs.3.rs-4244476. doi: 10.21203/rs.3.rs-4244476/v1.

Proteomic Profiling of SGLT-2 Inhibitor Canagliflozin in a Swine Model of Chronic Myocardial Ischemia.慢性心肌缺血猪模型中SGLT-2抑制剂卡格列净的蛋白质组学分析

Biomedicines. 2024 Mar 6;12(3):588. doi: 10.3390/biomedicines12030588.

DPP-4 inhibitor sitagliptin treatment results in altered myocardial metabolic proteome and oxidative phosphorylation in a swine model of chronic myocardial ischemia.在慢性心肌缺血猪模型中，二肽基肽酶-4（DPP-4）抑制剂西他列汀治疗导致心肌代谢蛋白质组和氧化磷酸化发生改变。

Physiol Rep. 2024 Mar;12(5):e15976. doi: 10.14814/phy2.15976.

Physiological myocardial F-FDG uptake pattern in oncologic PET/CT: comparison with findings in cardiac sarcoidosis.肿瘤PET/CT中心肌的生理性F-FDG摄取模式：与心脏结节病的表现比较

Asia Ocean J Nucl Med Biol. 2024;12(1):1-10. doi: 10.22038/AOJNMB.2023.70254.1490.

文献AI研究员

20分钟写一篇综述，助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型，支持多种主流文档格式。

立即体验