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豚鼠基底动脉平滑肌细胞中ATP代谢和细胞内镁离子对钙离子通道活性的调节作用

Modulation of Ca2+ channel activity by ATP metabolism and internal Mg2+ in guinea-pig basilar artery smooth muscle cells.

作者信息

McHugh D, Beech D J

机构信息

Department of Pharmacology, University of Leeds, Leeds, UK.

出版信息

J Physiol. 1996 Apr 15;492 ( Pt 2)(Pt 2):359-76. doi: 10.1113/jphysiol.1996.sp021314.

Abstract
  1. Single smooth muscle cells were isolated from the basilar artery of the guinea-pig and, within 10 h, inward currents through voltage-gated Ca2+ channels were recorded using the amphotericin or conventional whole-cell voltage-clamp techniques. 2. In amphotericin whole-cell recordings, bath application of 2,4-dinitrophenol (DNP, an uncoupler of mitochondrial ATP production) induced an initial stimulation (14% increase in 5 of 11 cells) and then pronounced inhibition (50% decrease in 9 of 11 cells within 9.5 min) of voltage-dependent Ca2+ current (I(Ca)) elicited by depolarizing to +10 mV in 1.5 mM extracellular Ca2+ solution. By contrast, inhibition of glycolysis by replacing glucose in the bath with 2-deoxy-D-glucose had no effect. 3. Na+ current through Ca2+ channels (I[(Ca)(Na)]) recorded in the absence of extracellular divalent cations also responded to DNP, again with stimulation followed by inhibition of current. The stimulation of I[(Ca)(Na)] was associated with a leftward shift of the Ca2+ channel activation curve which averaged -9 mV. A combination of 2-deoxy-D-glucose, mannoheptulose and 3-0-methyl-glucose had only minor effects on I[(Ca)(Na)], whereas rotenone had an effect similar to that of DNP in six of eight cells. 4. The amplitude of I[(Ca)(Na)] in conventional whole-cell recordings was not different from that in amphotericin whole-cell recordings, even without ATP in the recording pipette and with metabolic poisons in the bath solution. Furthermore, attempts to dephosphorylate the Ca2+ channels in ATP-free conditions did not prevent I[(Ca)(Na)], and a high concentration of Mg-ATP with or without a phosphorylation-supporting medium in the recording pipette did not increase its amplitude. 5. In the absence of ATP, Mg2+ inhibited whole-cell I[Ca)(Na)] with a K(d) of about 100 mu M at -10 mV and induced a leftward shift of the Ca2+ channel activation curve. When ATP and a phosphorylation-supporting medium were in the recording pipette the blocking effect of free Mg2+ was reduced but the shift in the Ca2+ channel activation curve was unaffected. 6. From these data it is suggested that inhibition of mitochondrial, but not glycolytic, ATP production has stimulatory and inhibitory effects on voltage-gated Ca2+ channels of basilar artery smooth muscle cells. Effects of intracellular Mg2+ on the Ca2+ channels were modulated by ATP and mimicked the effects of metabolic poisoning by DNP. A hypothesis is discussed in which the intracellular free Mg2+ concentration may be a key factor coupling ATP production to Ca2+ channels.
摘要
  1. 从豚鼠基底动脉分离出单个平滑肌细胞,并在10小时内,使用两性霉素或传统的全细胞电压钳技术记录通过电压门控Ca2+通道的内向电流。2. 在两性霉素全细胞记录中,浴槽中加入2,4-二硝基苯酚(DNP,线粒体ATP生成的解偶联剂),在1.5 mM细胞外Ca2+溶液中去极化至+10 mV诱发的电压依赖性Ca2+电流(I(Ca)),最初出现刺激(11个细胞中有5个增加14%),然后出现明显抑制(11个细胞中有9个在9.5分钟内减少50%)。相比之下,用2-脱氧-D-葡萄糖替换浴槽中的葡萄糖抑制糖酵解没有效果。3. 在无细胞外二价阳离子的情况下记录的通过Ca2+通道的Na+电流(I[(Ca)(Na)])也对DNP有反应,同样先是电流刺激然后是抑制。I[(Ca)(Na)]的刺激与Ca2+通道激活曲线向左移动相关,平均移动-9 mV。2-脱氧-D-葡萄糖、甘露庚酮糖和3-0-甲基葡萄糖的组合对I[(Ca)(Na)]只有轻微影响,而鱼藤酮在8个细胞中有6个细胞的作用与DNP相似。4. 传统全细胞记录中I[(Ca)(Na)]的幅度与两性霉素全细胞记录中的幅度没有差异,即使记录电极中没有ATP且浴槽溶液中有代谢毒物。此外,在无ATP条件下使Ca2+通道去磷酸化的尝试并不能阻止I[(Ca)(Na)],记录电极中有无磷酸化支持介质的高浓度Mg-ATP也不会增加其幅度。5. 在无ATP时,Mg2+在-10 mV时以约100 μM的解离常数抑制全细胞I[Ca)(Na)],并使Ca2+通道激活曲线向左移动。当记录电极中有ATP和磷酸化支持介质时,游离Mg2+的阻断作用减弱,但Ca2+通道激活曲线的移动不受影响。6. 从这些数据表明,抑制线粒体而非糖酵解的ATP生成对基底动脉平滑肌细胞的电压门控Ca2+通道有刺激和抑制作用。细胞内Mg2+对Ca2+通道的作用受ATP调节,并模拟了DNP代谢中毒的作用。讨论了一个假说,即细胞内游离Mg2+浓度可能是将ATP生成与Ca2+通道耦合的关键因素。

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