Soriano S, Banting G
Department of Biochemistry and BBSRC-funded Molecular Recognition Centre, University of Bristol, UK.
FEBS Lett. 1997 Feb 10;403(1):1-4. doi: 10.1016/s0014-5793(96)01516-5.
Some aspects of the roles of inositol trisphosphate (Insp3) and inositol tetrakisphosphate (Insp4) in Ca2+ homeostasis in terms of inositol trisphosphate 3-kinase B (IP3K-B) localization and activity are discussed. The model that we propose is also compatible with IP3K-B participating in the widely reported phenomenon of quantal release of Ca2+ from internal stores, at least in some biological systems.
本文讨论了三磷酸肌醇（Insp3）和四磷酸肌醇（Insp4）在通过三磷酸肌醇3-激酶B（IP3K-B）定位和活性维持钙离子稳态方面的作用。我们提出的模型也与IP3K-B参与广泛报道的从内部储存库中量子式释放钙离子的现象相符，至少在某些生物系统中是这样。
