高血糖、腰丛神经病和低钾性横纹肌溶解症并发原发性醛固酮增多症。
Hyperglycemia, lumbar plexopathy and hypokalemic rhabdomyolysis complicating Conn's syndrome.
作者信息
Chow C P, Symonds C J, Zochodne D W
机构信息
Department of Clinical Neurosciences, University of Calgary, Alberta, Canada.
出版信息
Can J Neurol Sci. 1997 Feb;24(1):67-9. doi: 10.1017/s0317167100021132.
BACKGROUND
Lumbosacral plexopathy is a complication of diabetes mellitus. Conn's syndrome from an aldosterone secreting adenoma may be associated with hypokalemia and rhabdomyolysis but mild hyperglycemia also usually occurs.
METHODS
Case description.
RESULTS
A 70-year-old male diagnosed as having Conn's syndrome, hypokalemia and mild hyperglycemia developed rhabdomyolysis and lumbar plexopathy as a presenting feature of his hyperaldosteronism. His rhabdomyolysis rapidly cleared following correction of hypokalemia but recovery from the plexopathy occurred slowly over several months. Definite resection of the aldosterone secreting adenomas reversed the hyperglycemia.
CONCLUSIONS
Our patient developed lumbar plexopathy resembling that associated with diabetes mellitus despite the presence of only mild and transient hyperglycemia.
背景
腰骶丛神经病是糖尿病的一种并发症。醛固酮分泌腺瘤引起的Conn综合征可能与低钾血症和横纹肌溶解有关，但通常也会出现轻度高血糖。
方法
病例描述。
结果
一名70岁男性被诊断为Conn综合征、低钾血症和轻度高血糖，出现横纹肌溶解和腰骶丛神经病，作为其醛固酮增多症的首发特征。低钾血症纠正后，他的横纹肌溶解迅速清除，但丛神经病在数月内缓慢恢复。醛固酮分泌腺瘤的明确切除逆转了高血糖。
结论
尽管仅存在轻度和短暂的高血糖，我们的患者仍出现了类似于糖尿病相关的腰骶丛神经病。
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