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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Fuchi I, Higashino H, Noda K, Suzuki A, Matsubara Y

Department of Obstetrics and Gynecology, Kinki University School of Medicine, Osaka-Sayama, Japan.

Clin Exp Pharmacol Physiol Suppl. 1995 Dec;22(1):S283-5. doi: 10.1111/j.1440-1681.1995.tb02919.x.

Placental Na+, K+ -ATPase activities were measured in connection with the active transport of amino acid, intra-uterine growth retardation (IUGR) or small for date infants (SFD) in salt-loaded pregnant SHRSP. 2. All parameters such as fetal weight, placental weight and Na+, K+ -ATPase activity in the placental tissue were significantly depressed in SHRSP, especially in the salt-loaded SHRSP compared with WKY. 3. Besides a decrease of blood flow, a decline of Na+, K+ -ATPase activity associated with an active transport of amino acid in the placenta would cause IUGR and SFD in the salt-loaded pregnant SHRSP. 4. Pregnant SHRSP given water containing salt would be a good model for pregnancy induced hypertension in humans.

在盐负荷的妊娠自发性高血压大鼠（SHRSP）中，测量胎盘钠钾ATP酶活性，并将其与氨基酸的主动转运、宫内生长迟缓（IUGR）或小于胎龄儿（SFD）相关联。2. 与WKY大鼠相比，SHRSP大鼠的所有参数，如胎儿体重、胎盘重量和胎盘组织中的钠钾ATP酶活性均显著降低，尤其是盐负荷的SHRSP大鼠。3. 除了血流量减少外，与胎盘氨基酸主动转运相关的钠钾ATP酶活性下降会导致盐负荷妊娠SHRSP大鼠出现IUGR和SFD。4. 给予含盐饮水的妊娠SHRSP大鼠将是人类妊娠高血压的良好模型。

Fuchi I, Higashino H, Noda K, Suzuki A, Matsubara Y

Department of Obstetrics and Gynecology, Kinki University School of Medicine, Osaka-Sayama, Japan.

Clin Exp Pharmacol Physiol Suppl. 1995 Dec;22(1):S283-5. doi: 10.1111/j.1440-1681.1995.tb02919.x.

Placental Na+, K+ -ATPase activities were measured in connection with the active transport of amino acid, intra-uterine growth retardation (IUGR) or small for date infants (SFD) in salt-loaded pregnant SHRSP. 2. All parameters such as fetal weight, placental weight and Na+, K+ -ATPase activity in the placental tissue were significantly depressed in SHRSP, especially in the salt-loaded SHRSP compared with WKY. 3. Besides a decrease of blood flow, a decline of Na+, K+ -ATPase activity associated with an active transport of amino acid in the placenta would cause IUGR and SFD in the salt-loaded pregnant SHRSP. 4. Pregnant SHRSP given water containing salt would be a good model for pregnancy induced hypertension in humans.

在盐负荷的妊娠自发性高血压大鼠（SHRSP）中，测量胎盘钠钾ATP酶活性，并将其与氨基酸的主动转运、宫内生长迟缓（IUGR）或小于胎龄儿（SFD）相关联。2. 与WKY大鼠相比，SHRSP大鼠的所有参数，如胎儿体重、胎盘重量和胎盘组织中的钠钾ATP酶活性均显著降低，尤其是盐负荷的SHRSP大鼠。3. 除了血流量减少外，与胎盘氨基酸主动转运相关的钠钾ATP酶活性下降会导致盐负荷妊娠SHRSP大鼠出现IUGR和SFD。4. 给予含盐饮水的妊娠SHRSP大鼠将是人类妊娠高血压的良好模型。