Rogers I M
South Tyneside Health Care Trust, South Shields, Tyne and Wear, UK.
Acta Paediatr. 1997 Jan;86(1):6-9. doi: 10.1111/j.1651-2227.1997.tb08821.x.
A theory is advanced about the cause of pyloric stenosis of infancy (PS). Developmental changes will conspire to produce pathogenetic gastric hyperacidity within the first 4 weeks of life in babies who develop PS. The prime cause will be an increased gastric acidity due to a genetically determined supernormal parietal cell mass. This theory satisfactorily explains many known clinical features.
关于婴儿期幽门狭窄(PS)的病因提出了一种理论。在患PS的婴儿出生后的前4周内,发育变化共同作用会导致致病性胃酸过多。主要原因将是由于基因决定的超常壁细胞数量导致胃酸增加。该理论令人满意地解释了许多已知的临床特征。