Williams K R, Blayney A W, Lesser T H
Department of Basic Dental Science, Dental School, University of Wales College of Medicine, Cardiff, UK.
Clin Otolaryngol Allied Sci. 1997 Apr;22(2):126-31. doi: 10.1046/j.1365-2273.1997.00874.x.
This work makes use of recent finite element discretization of the human tympanic membrane where the known anisotropy of the tympanic membrane is implemented by a series of thin beam elements superimposed on thin shell membrane elements. These thin beam elements were introduced in order to simulate the fibre structure of the tympanic membrane. The onset of tympanosclerosis has been modelled as a gradual increase in tympanic membrane density and modulus in the affected area. This increased density results in a fall of 55% of the first natural frequency for only a 16% area of drum sclerosis. Additionally the amplitude response is significantly reduced. Repair of the membrane was simulated by removal of the fibre system from the affected area. Under these circumstances the natural frequencies virtually return to the healthy state but the mode shapes of vibration do not. The repair to the membrane, although not significantly altering frequency values, has resulted in a disruption to the normal mode shape patterns with consequences in the movements conveyed to the stapes and cochlear fluids.
这项工作利用了近期对人鼓膜的有限元离散化方法,其中通过叠加在薄壳膜单元上的一系列细梁单元来实现已知的鼓膜各向异性。引入这些细梁单元是为了模拟鼓膜的纤维结构。鼓室硬化症的发病已被建模为受影响区域鼓膜密度和模量的逐渐增加。仅16%的鼓膜硬化区域,这种密度增加就导致第一固有频率下降55%。此外,振幅响应也显著降低。通过从受影响区域去除纤维系统来模拟鼓膜修复。在这种情况下,固有频率实际上恢复到健康状态,但振动模态形状并未恢复。鼓膜修复虽然没有显著改变频率值,但却导致了正常模态形状模式的破坏,进而影响了传递到镫骨和耳蜗液的运动。