Chistiakova M A, Grushina A G
Zh Nevropatol Psikhiatr Im S S Korsakova. 1977;77(9):1309-14.
The paper deals with the possible mechanism in the development of a hemorrhagic brain infarction during myocardial infarction, complicated by a cardiogenic shock of the III degree. The results of a postmortal study of the brain, its major arteries, as well as the vessels of the Willis circle, permit to assume that the mechanism of realization of this cerebral vascular complication is in a joint metabolic and microcirculatory disorders accompanying the shock. Besides, there is also a stenosing atherosclerotic process, with prevalent lesion of the intracranial part of the magistral arteries, supplying the brain.
本文探讨了心肌梗死并发Ⅲ度心源性休克时发生出血性脑梗死的可能机制。对脑、其主要动脉以及Willis环血管的尸检研究结果表明，这种脑血管并发症的发生机制在于休克伴随的联合代谢和微循环障碍。此外，还存在动脉粥样硬化狭窄过程，主要累及供应脑的颅内部分主要动脉。
