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Modulation of clinical expression and band 3 deficiency in hereditary spherocytosis.

作者信息

Alloisio N, Texier P, Vallier A, Ribeiro M L, Morlé L, Bozon M, Bursaux E, Maillet P, Gonçalves P, Tanner M J, Tamagnini G, Delaunay J

机构信息

Laboratoire de Génétique Moléculaire Humaine, Centre National de la Recherche Scientifique, URA 1171, Institut Pasteur de Lyon, France.

出版信息

Blood. 1997 Jul 1;90(1):414-20.

PMID:9207478
Abstract

We present two novel alleles of the anion-exchanger 1 (AE1) gene, allele Coimbra and allele Mondego. Allele Coimbra (V488M, GTG --> ATG) affects a conserved position in the putative second ectoplasmic loop of erythrocyte band 3. In 15 simple heterozygotes, it yielded a mild form of hereditary spherocytosis (HS) with band 3 deficiency (-20% +/- 2%) and a reduced number of 4,4'-diisothiocyano-1,2-diphenylethane-2,2'-disulfonate (H2DIDS) binding sites (-35%). However, two additional heterozygotes presented with an aggravated HS and a more pronounced reduction of band 3 (-40%) and of H2DIDS binding sites (-48%). They carried, in trans to allele Coimbra, allele Mondego, defined by two mutations: E40K, GAG --> AAG, the known mutation Montefiore, and P147S, CCT --> TCT, a novel mutation, both located in the cytoplasmic domain of band 3. Allele Mondego itself resulted in no clinical or hematologic HS signs in the simple heterozygous state. Yet it yielded a slight decrease in band 3 (-6% to -12%) and in the number of H2DIDS binding sites (-19%). Thus, the more pronounced decrease in band 3 in the two compound heterozygotes derived from the additive effects of two unequally expressed AE1 alleles, resulting in a more severe clinical picture.

摘要

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