Bulygin I A, Reprintseva V M
Fiziol Zh SSSR Im I M Sechenova. 1977 Oct;63(10):1438-45.
Adrenaline (A) and noradrenaline (N) release from the caudal mesenteric ganglion (CMG) during preganglionic nerve stimulation is due to acetylcholine action on N--and M-cholinoreceptors of ganglion's adrenergic structures (GAS) and probably, primarily on SIF--cells, whereas during peripheral reflexes of CMG it is related to the excitation of noncholinergic afferent neurons which affect the corresponding receptors of GAS. A and N release is inhibited via M-receptors. There is a complex relationship between receptor fields of GAS. Hemicholinium as well as N--cholinoblocking agents, during first minutes of their application to the ganglion elicit A and N release both before and after preganglionic fiber degeneration, which indicates their postsynaptic effect on the cholinoreceptors. Changes in A and N in the ganglion during peripheral reflexes are increased after prolonged action of hemicholinium and chronic decentralisation of CMG.
在节前神经刺激期间，来自肠系膜后神经节（CMG）的肾上腺素（A）和去甲肾上腺素（N）释放是由于乙酰胆碱作用于神经节肾上腺素能结构（GAS）的N - 和M - 胆碱能受体，可能主要作用于SIF细胞；而在CMG的外周反射期间，它与非胆碱能传入神经元的兴奋有关，这些神经元影响GAS的相应受体。A和N的释放通过M受体被抑制。GAS的受体场之间存在复杂的关系。半胆碱以及N - 胆碱阻断剂在应用于神经节的最初几分钟内，在节前纤维变性前后都会引起A和N的释放，这表明它们对胆碱能受体有突触后效应。在半胆碱的长时间作用和CMG的慢性去传入后，外周反射期间神经节中A和N的变化会增加。
