A hypothesis that aging results from defects in genetically produced proteins.
作者信息
Secerbegovic S
机构信息
Department of Biology, University of Sarajevo, Bosnia.
出版信息
Med Hypotheses. 1997 Jun;48(6):531-3. doi: 10.1016/s0306-9877(97)90125-0.
PMID:9247899
Abstract
The hypothesis is presented that our life span is genetically programmed and that it is possible that a defect in produced proteins encoded by the 'longevity' gene is a cause of aging. A study of correlations between defective enzyme activity in two diseases characterized by premature aging (progeria and Werner's syndrome) could assist in elucidating the common mechanism for all aging processes.
摘要
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