Iesaka Y, Takahashi A, Goya M, Soejima Y, Okamoto Y, Fujiwara H, Aonuma K, Nogami A, Hiroe M, Marumo F, Hiraoka M
Cardiovascular Center, Tsuchiura Kyodo Hospital, Ibaraki-ken, Japan.
J Cardiovasc Electrophysiol. 1997 Aug;8(8):854-64. doi: 10.1111/j.1540-8167.1997.tb00846.x.
Atrial tachycardia shows wide variations in its electrophysiologic properties and sites of origin. We report an atrial tachycardia with ECG manifestations and electrophysiologic characteristics similar to an atypical form of AV nodal reentrant tachycardia (AVNRT).
This supraventricular tachycardia was observed in 11 patients. It was initiated by atrial extrastimulation with an inverse relationship between the coupling interval of an extrastimulus and the postextrastimulus interval. Its induction was not related to a jump in the AH interval, and its perpetuation was independent of conduction block in AV node. Ventricular pacing during tachycardia demonstrated AV dissociation without affecting the atrial cycle length. A very small dose of adenosine triphosphate (mean 3.9 +/- 1.2 mg) could terminate the tachycardia. The earliest atrial activation during tachycardia was recorded at the low anteroseptal right atrium with a different intra-atrial activation sequence from that recorded during ventricular pacing, where the tachycardia was successfully ablated in 9 of 10 attempted patients. Bidirectional AV nodal conduction remained unaffected after successful ablation.
There may be an entity of adenosine-sensitive atrial tachycardia probably due to focal reentry within the AV node or its transitional tissues without involvement of the AV nodal pathways. This tachycardia can be ablated without disturbing AV nodal conduction from the right atrial septum.
房性心动过速在电生理特性和起源部位上表现出广泛的差异。我们报告了一种房性心动过速,其心电图表现和电生理特征类似于非典型房室结折返性心动过速(AVNRT)。
在11例患者中观察到这种室上性心动过速。它由心房期外刺激诱发,期外刺激的耦合间期与期外刺激后间期呈反比关系。其诱发与AH间期的跳跃无关,其持续存在与房室结传导阻滞无关。心动过速期间的心室起搏显示房室分离,而不影响心房周期长度。极少量的三磷酸腺苷(平均3.9±1.2毫克)即可终止心动过速。心动过速期间最早的心房激动记录于低位前间隔右心房,其心房内激动顺序与心室起搏时记录的不同,在10例尝试消融的患者中有9例在此成功消融了心动过速。成功消融后双向房室结传导仍未受影响。
可能存在一种对腺苷敏感的房性心动过速,可能是由于房室结或其过渡组织内的局灶性折返,而未累及房室结径路。这种心动过速可从右心房隔部进行消融,而不干扰房室结传导。