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腺苷敏感性结周房性心动过速。其机制是什么？

Adenosine-sensitive perinodal atrial tachycardia. What is the mechanism?

作者信息

Vijay Soorampally, Patodia Ansul, Lokhandwala Yash

机构信息

Department of Cardiology, Holy Family Hospital, Mumbai, India.

Department of Cardiology, Holy Family Hospital, Mumbai, India.

出版信息

Indian Pacing Electrophysiol J. 2023 May-Jun;23(3):91-93. doi: 10.1016/j.ipej.2023.03.001. Epub 2023 Mar 5.

DOI:10.1016/j.ipej.2023.03.001
PMID:36871891
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC10160748/
Abstract

Termination of focal atrial tachycardia with adenosine is considered a defining feature for triggered activity. Recent evidence, however, suggests that the perinodal adenosine-sensitive AT has reentry as the mechanism of tachycardia. In this report, we were able to confirm the mechanism of AT as reentry by observing the response to programmed electrical stimulation and demonstrating the fallacy of traditional teaching that the adenosine responsiveness of AT is a criterion for labeling the mechanism as triggered activity.

摘要

腺苷终止局灶性房性心动过速被认为是触发活动的一个决定性特征。然而，最近的证据表明，结周腺苷敏感的房性心动过速的机制是折返。在本报告中，我们通过观察程序性电刺激的反应，并证明传统观点（即房性心动过速的腺苷反应性是将机制标记为触发活动的标准）的错误，能够确认房性心动过速的机制是折返。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0609/10160748/4d9e9ba40b41/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0609/10160748/8cc915bebe27/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0609/10160748/4d9e9ba40b41/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0609/10160748/8cc915bebe27/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0609/10160748/4d9e9ba40b41/gr2.jpg

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本文引用的文献

1
Reappraisal and new observations on atrial tachycardia ablated from the non-coronary aortic sinus of Valsalva.对源自主动脉窦的非冠状动脉的房性心动过速的再评估和新观察。
Europace. 2018 Jan 1;20(1):124-133. doi: 10.1093/europace/euw324.
2
Adenosine-insensitive focal atrial tachycardia: evidence for de novo micro-re-entry in the human atrium.腺苷不敏感型局灶性房性心动过速：人体心房新发生微折返的证据。
J Am Coll Cardiol. 2007 Mar 27;49(12):1324-33. doi: 10.1016/j.jacc.2006.11.037. Epub 2007 Mar 9.
3
Adenosine-sensitive atrial reentrant tachycardia originating from the atrioventricular nodal transitional area.
起源于房室结过渡区的腺苷敏感性房性折返性心动过速。
J Cardiovasc Electrophysiol. 1997 Aug;8(8):854-64. doi: 10.1111/j.1540-8167.1997.tb00846.x.
4
Is the response to programmed electrical stimulation diagnostic of mechanisms for arrhythmias?程序性电刺激的反应能否诊断心律失常的机制？
Circulation. 1986 Feb;73(2 Pt 2):II18-27.