Campbell P J, Edwards S M
University of California, Davis Medical Center, Neurosurgical Intensive Care Unit, Sacramento 95817, USA.
J Neurosci Nurs. 1997 Oct;29(5):318-24. doi: 10.1097/01376517-199710000-00005.
The onset of subtle diffuse ischemic neurological deficits often associated with cerebral vasospasm is a major cause of morbidity and mortality following aneurysmal subarachnoid hemorrhage. The exact etiology of cerebral vasospasm is unclear. Increasing intravascular volume, decreasing blood viscosity and inducing hypertension may help prevent or diminish neurological deficits from cerebral vasospasm by improving cerebral blood flow. An intensive multidisciplinary approach is necessary with the role of the neuroscience nurse being pivotal. An understanding of the subtle neurological changes suggestive of cerebral vasospasm and its effects leads to early recognition, and allows for rapid institution of therapy.
与脑血管痉挛相关的细微弥漫性缺血性神经功能缺损的出现是动脉瘤性蛛网膜下腔出血后发病和死亡的主要原因。脑血管痉挛的确切病因尚不清楚。增加血管内容量、降低血液粘度和诱导高血压可能通过改善脑血流有助于预防或减轻脑血管痉挛引起的神经功能缺损。必须采取强化的多学科方法,神经科学护士的作用至关重要。了解提示脑血管痉挛的细微神经变化及其影响可导致早期识别,并能迅速开始治疗。