Krämer K D, Kult J, Hochrein H
Basic Res Cardiol. 1976 May-Jun;71(3):270-86. doi: 10.1007/BF01906452.
In tests on guinea pig hearts in situ asphyxia tolerance was found to be increased early in hemorragic shock four times control value. These results are suggestive of an energy saving mechanism in shock whereby the heart is not damaged out metabolically relieved. Myocardial analyses showed that, owing to shock-related cardiac relief, under a simultaneous asphyctic stress the intracellular potassium stock decreases later and markedly less.
在对豚鼠心脏进行的原位测试中发现，在失血性休克早期，心脏的窒息耐受性增加至对照值的四倍。这些结果表明休克中存在一种节能机制，由此心脏不会因代谢缓解而受损。心肌分析显示，由于休克相关的心脏缓解作用，在同时存在窒息应激的情况下，细胞内钾储备下降得更晚且明显更少。
