Hirose Y, Ishida Y, Hayashida K, Satoh T, Shimotsu Y, Nishimura T
Department of Radiology, National Cardiovascular Center, Osaka, Japan.
Cardiology. 1997 Nov-Dec;88(6):609-12. doi: 10.1159/000177435.
We report on a 47-year-old woman who experienced an Eisenmenger reaction (mean pulmonary artery pressure: 86 mm Hg) induced by atrial septal defect. Radionuclide myocardial scans with 99mTc-MIBI and 123I-BMIPP showed increased uptake to the right ventricular myocardium, whereas the 123I-MIBG scan disclosed no uptake to the right ventricular myocardium. The scans showed no apparent abnormality in the left ventricular myocardium. These findings suggest that the right ventricular myocardium was viable but denervated due to severe pulmonary hypertension. The mechanism of right ventricular failure may be closely related to sympathetic denervation.
我们报告了一名47岁女性，她因房间隔缺损引发了艾森曼格反应（平均肺动脉压：86 mmHg）。用99mTc-MIBI和123I-BMIPP进行的放射性核素心肌扫描显示右心室心肌摄取增加，而123I-MIBG扫描显示右心室心肌无摄取。扫描显示左心室心肌无明显异常。这些发现表明右心室心肌存活但因严重肺动脉高压而失神经支配。右心室衰竭的机制可能与交感神经失神经支配密切相关。
