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"Invasive-growth" signaling by the Met/HGF receptor: the hereditary renal carcinoma connection.

作者信息

Bardelli A, Pugliese L, Comoglio P M

机构信息

Institute for Cancer Research (IRCC), University of Torino Medical School, Candiolo, Turin, Italy.

出版信息

Biochim Biophys Acta. 1997 Dec 9;1333(3):M41-51. doi: 10.1016/s0304-419x(97)00026-7.

DOI:10.1016/s0304-419x(97)00026-7
PMID:9426201
Abstract
摘要

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"Invasive-growth" signaling by the Met/HGF receptor: the hereditary renal carcinoma connection.
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Control of invasive growth by the HGF receptor family.HGF受体家族对侵袭性生长的调控
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The hepatocyte growth factor receptor as a potential therapeutic target for dedifferentiated liposarcoma.肝细胞生长因子受体作为去分化脂肪肉瘤的潜在治疗靶点。
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Hepatocyte growth factor-induced Ras activation requires ERM proteins linked to both CD44v6 and F-actin.肝细胞生长因子诱导的Ras激活需要与CD44v6和F-肌动蛋白相连的ERM蛋白。
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Activation of cdc42, rac, PAK, and rho-kinase in response to hepatocyte growth factor differentially regulates epithelial cell colony spreading and dissociation.响应肝细胞生长因子时，细胞分裂周期蛋白42（cdc42）、Rac蛋白、p21激活激酶（PAK）和 Rho激酶的激活对上皮细胞集落的扩散和解离具有不同的调节作用。
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Uncoupling signal transducers from oncogenic MET mutants abrogates cell transformation and inhibits invasive growth.使致癌性MET突变体的信号转导器解偶联可消除细胞转化并抑制侵袭性生长。
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Protein tyrosine phosphatase PTP-S binds to the juxtamembrane region of the hepatocyte growth factor receptor Met.蛋白酪氨酸磷酸酶PTP-S与肝细胞生长因子受体Met的近膜区域结合。
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