Lagier R, Van Linthoudt D
Int Orthop. 1979;3(1):1-8. doi: 10.1007/BF00266319.
Two cases of Sudeck's atrophy of the foot occurring 16 months and 17 years after infected fractures of the leg were studied radiologically and pathologically. Various cartilaginous changes were observed, the nature and severity of which depended on the joint involved. They included superficial pannus, deep erosion, fibrous ankylosis and, at times, bony ankylosis. These changes are similar to those observed in nine cases studied by Rutishauser et al. Comparison of the pathological changes in human and experimental joint immobilization suggests that these changes are due mainly to decreased mobility of the joints of the foot in Sudeck's atrophy. These observations also suggest that physiotherapeutic mobilization in Sudeck's atrophy is important for the joints as well as for bone. From a more general point of view, they demonstrate that a condition which is nosologically different from the chronic rheumatic diseases can nevertheless cause lesions that are a fundamental part of the pathological changes in osteoarthritis, rheumatoid arthritis and ankylosing spondylitis.
对两例分别在腿部感染性骨折后16个月和17年出现足部苏戴克萎缩的病例进行了放射学和病理学研究。观察到各种软骨变化,其性质和严重程度取决于受累关节。这些变化包括浅表血管翳、深部侵蚀、纤维性强直,有时还有骨性强直。这些变化与鲁蒂沙伊泽等人研究的9例病例中观察到的变化相似。对人体和实验性关节固定的病理变化进行比较表明,这些变化主要是由于苏戴克萎缩中足部关节活动度降低所致。这些观察结果还表明,苏戴克萎缩中的物理治疗性活动对关节和骨骼都很重要。从更普遍的角度来看,它们表明一种在病因学上与慢性风湿性疾病不同的病症,仍然可以引起作为骨关节炎、类风湿性关节炎和强直性脊柱炎病理变化基本组成部分的病变。