Berger R, Jacobs W
Pacing Clin Electrophysiol. 1979 Nov;2(6):596-602. doi: 10.1111/j.1540-8159.1979.tb04278.x.
Myopotential inhibition of demand pacemakers is a common phenomenon and is responsible for a greater degree of morbidity and possible mortality in patients with pacers. Significant electrical potentials generated by contracting muscle mass gain entry into the pacemaker circuitry causing inhibition of pacemaker activity. Intraoperative and bedside maneuvers can readily precipitate the problem and allow for accurate diagnosis. Holter monitoring frequently can also serve to establish the diagnosis. Pacemaker repositioning, replacement with a bipolar system, use of a fixed-rate pacemaker, and pacemaker "sleeves" are possible intraoperative therapeutic methods. External sensitivity adjustable pacemakers would seemingly allow for noninvasive solutions to the problem.
需求型起搏器的肌电位抑制是一种常见现象，并且是起搏器患者中更高发病率和可能死亡率的原因。收缩的肌肉团块产生的显著电位进入起搏器电路，导致起搏器活动受到抑制。术中及床边操作很容易引发该问题并实现准确诊断。动态心电图监测通常也有助于确诊。起搏器重新定位、更换为双极系统、使用固定频率起搏器以及起搏器“套管”是可能的术中治疗方法。外部灵敏度可调起搏器似乎可为该问题提供无创解决方案。
