Acasuso-Díaz M, Collantes-Estévez E
Sant Jordi Centro Médico Reus, Tarragona, Spain.
Arthritis Care Res. 1998 Feb;11(1):39-42. doi: 10.1002/art.1790110107.
To test the hypothesis that joint hyperlaxity can play some role in the pathogenesis of pain in primary fibromyalgia.
A total of 66 women with fibromyalgia (according to the 1990 American College of Rheumatology criteria) and 70 women with other rheumatic diseases were examined for joint laxity based on 5 criteria (The Non-Dominant Spanish modification). Individuals meeting 4 or 5 criteria were considered to be hyperlax.
Joint hyperlaxity was detected in 18 (27.3%) of the patients with fibromyalgia and 8 (11.4%) of those with another rheumatic disorder. The statistical analysis revealed significant differences (P < 0.05) between both groups.
The results of this study suggest that joint hypermobility and fibromyalgia are associated. Joint hyperlaxity may play a prominent role in the pathogenesis of pain in fibromyalgia.
检验关节过度松弛在原发性纤维肌痛疼痛发病机制中可能起一定作用这一假设。
根据5项标准（非优势侧西班牙改良版），对66名患有纤维肌痛的女性（根据1990年美国风湿病学会标准）和70名患有其他风湿性疾病的女性进行关节松弛度检查。符合4项或5项标准的个体被认为是关节过度松弛。
在患有纤维肌痛的患者中，18人（27.3%）检测出关节过度松弛，在患有其他风湿性疾病的患者中，8人（11.4%）检测出关节过度松弛。统计分析显示两组之间存在显著差异（P < 0.05）。
本研究结果表明关节活动过度与纤维肌痛有关。关节过度松弛可能在纤维肌痛疼痛的发病机制中起重要作用。
