Immunopathology of apoptosis--introduction and overview.

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作者信息

Vaux D L

机构信息

Walter and Eliza Hall Institute for Medical Research, Post Office Royal Melbourne Hospital, Victoria, Australia.

出版信息

Springer Semin Immunopathol. 1998;19(3):271-8. doi: 10.1007/BF00787224.

DOI:10.1007/BF00787224

PMID:9540156

Abstract

摘要

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本文引用的文献

The role of the bcl-2/ced-9 gene family in cancer and general implications of defects in cell death control for tumourigenesis and resistance to chemotherapy.bcl-2/ced-9基因家族在癌症中的作用以及细胞死亡调控缺陷对肿瘤发生和化疗耐药性的一般影响。

Biochim Biophys Acta. 1997 Oct 24;1333(2):F151-78. doi: 10.1016/s0304-419x(97)00019-x.

Caspases: killer proteases.半胱天冬酶：杀手蛋白酶。

Trends Biochem Sci. 1997 Aug;22(8):299-306. doi: 10.1016/s0968-0004(97)01085-2.

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3.Apaf-1是一种与秀丽隐杆线虫CED-4同源的人类蛋白质，参与细胞色素c依赖性的半胱天冬酶-3激活过程。

Cell. 1997 Aug 8;90(3):405-13. doi: 10.1016/s0092-8674(00)80501-2.

The regulation of anoikis: MEKK-1 activation requires cleavage by caspases.失巢凋亡的调控：MEKK-1的激活需要半胱天冬酶进行切割。

Cell. 1997 Jul 25;90(2):315-23. doi: 10.1016/s0092-8674(00)80339-6.

X-linked IAP is a direct inhibitor of cell-death proteases.X连锁凋亡抑制蛋白是细胞死亡蛋白酶的直接抑制剂。

Nature. 1997 Jul 17;388(6639):300-4. doi: 10.1038/40901.

A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis.一种组合方法确定了半胱天冬酶家族成员和颗粒酶B的特异性。为细胞凋亡的关键介质建立了功能关系。

J Biol Chem. 1997 Jul 18;272(29):17907-11. doi: 10.1074/jbc.272.29.17907.

Inhibition of death receptor signals by cellular FLIP.细胞型FLIP对死亡受体信号的抑制作用。

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Bcl-2 rescues T lymphopoiesis in interleukin-7 receptor-deficient mice.Bcl-2挽救白细胞介素-7受体缺陷小鼠的T淋巴细胞生成。

Cell. 1997 Jun 27;89(7):1033-41. doi: 10.1016/s0092-8674(00)80291-3.

Bcl-2 can rescue T lymphocyte development in interleukin-7 receptor-deficient mice but not in mutant rag-1-/- mice.Bcl-2可挽救白细胞介素-7受体缺陷小鼠的T淋巴细胞发育，但不能挽救突变型rag-1-/-小鼠的T淋巴细胞发育。

Cell. 1997 Jun 27;89(7):1011-9. doi: 10.1016/s0092-8674(00)80289-5.

Caenorhabditis elegans CED-4 stimulates CED-3 processing and CED-3-induced apoptosis.秀丽隐杆线虫的CED-4刺激CED-3的加工以及CED-3诱导的细胞凋亡。

Curr Biol. 1997 Jul 1;7(7):455-60. doi: 10.1016/s0960-9822(06)00216-8.

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