Kartashev I P, Titov V V
Fiziol Zh SSSR Im I M Sechenova. 1976 Jul;62(7):1062-7.
When breathing with air, pO2 in anesthetized rats was 24.1+/-1.8 mm Hg, while after urea administration it was 19.4+/-1.7 mm Hg. A sharp increase in pO2 was revealed in hyperoxia(911+/-48 mm Hg at 6 atm during first 5 min, and 924+/-38 mm Hg--by the 7th min). The high pO2 was only reduced in 10-15 min after decompression. At a combined action of urea and hyperoxia, pO2 was significantly lower (714+/-52 mm Hg); pO2 in successive cortical layers (0-2400 mcm with a 200-mcm step) was progressively decreasing (from 40.7+/-4.9 mm Hg to 16.0+/-1.0 mm Hg in control, and from 928+/-35 mm Hg to 274+/-64 mm Hg in hyperoxia).
吸入空气时，麻醉大鼠的动脉血氧分压（pO2）为24.1±1.8毫米汞柱，而给予尿素后为19.4±1.7毫米汞柱。在高氧环境中，pO2急剧升高（最初5分钟内在6个大气压下为911±48毫米汞柱，到第7分钟时为924±38毫米汞柱）。减压后10 - 15分钟，高pO2才开始下降。在尿素和高氧联合作用下，pO2显著降低（714±52毫米汞柱）；连续皮质层（0 - 2400微米，步长200微米）中的pO2逐渐降低（对照组从40.7±4.9毫米汞柱降至16.0±1.0毫米汞柱，高氧环境中从928±35毫米汞柱降至274±64毫米汞柱）。
