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PROTEIN-CARBOHYDRATE INTERACTION. II. INHIBITION STUDIES ON THE INTERACTION OF CONCANAVALIN A WITH POLYSACCHARIDES.蛋白质-碳水化合物相互作用。II. 伴刀豆球蛋白A与多糖相互作用的抑制研究。
Biochemistry. 1965 May;4:876-83. doi: 10.1021/bi00881a013.
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Specificity determinants in neurotrophin-3 and design of nerve growth factor-based trkC agonists by changing central beta-strand bundle residues to their neurotrophin-3 analogs.神经营养因子-3中的特异性决定因素以及通过将中枢β-链束残基替换为其神经营养因子-3类似物来设计基于神经生长因子的trkC激动剂。
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Two restricted sites on the surface of the nerve growth factor molecule independently determine specific TrkA receptor binding and activation.神经生长因子分子表面的两个受限位点独立决定了特定TrkA受体的结合与激活。
J Biol Chem. 1997 Apr 4;272(14):9300-7. doi: 10.1074/jbc.272.14.9300.
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Kinetic analysis of biosensor data: elementary tests for self-consistency.生物传感器数据的动力学分析:自洽性的基本测试
Trends Biochem Sci. 1996 Dec;21(12):458-60. doi: 10.1016/s0968-0004(96)20025-8.
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The neurotrophins and CNTF: two families of collaborative neurotrophic factors.神经营养因子与睫状神经营养因子:两类协同作用的神经营养因子家族。
Annu Rev Neurosci. 1996;19:491-515. doi: 10.1146/annurev.ne.19.030196.002423.
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Deletions in the extracellular domain of rat trkA lead to an altered differentiative phenotype in neurotrophin responsive cells.大鼠trkA细胞外结构域的缺失导致神经营养因子反应性细胞的分化表型改变。
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Characterization of histidine residues essential for receptor binding and activity of nerve growth factor.对神经生长因子受体结合及活性至关重要的组氨酸残基的特性研究
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8
Dimerization of the extracellular domain of the erythropoietin (EPO) receptor by EPO: one high-affinity and one low-affinity interaction.促红细胞生成素(EPO)使促红细胞生成素受体的细胞外结构域二聚化:一种高亲和力相互作用和一种低亲和力相互作用。
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9
The interleukin-6 (IL-6) partial antagonist (Q159E,T162P)IL-6 interacts with the IL-6 receptor and gp130 but fails to induce a stable hexameric receptor complex.白细胞介素-6(IL-6)部分拮抗剂(Q159E,T162P)IL-6与IL-6受体和gp130相互作用,但无法诱导形成稳定的六聚体受体复合物。
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Binding in the growth hormone receptor complex.生长激素受体复合物中的结合。
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神经营养因子受体TrkA重组细胞外结构域的特性及其与神经生长因子(NGF)的相互作用。

Characterization of the recombinant extracellular domain of the neurotrophin receptor TrkA and its interaction with nerve growth factor (NGF).

作者信息

Woo S B, Whalen C, Neet K E

机构信息

Department of Biological Chemistry, Finch University of Health Sciences/The Chicago Medical School, Illinois 60064, USA.

出版信息

Protein Sci. 1998 Apr;7(4):1006-16. doi: 10.1002/pro.5560070418.

DOI:10.1002/pro.5560070418
PMID:9568907
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC2143976/
Abstract

Nerve growth factor (NGF) is the prototype of a family of neurotrophins that support important neuronal programs such as differentiation and survival of a subset of sympathetic, sensory, and brain neurons. NGF binds to two classes of cell surface receptors: p75LANR and p140TrkA. NGF binding to p140TrkA initiates the neuronal signaling pathway through activation of the tyrosine kinase activity, which subsequently results in a rapid signal transduction through a phosphorylation cascade. To examine this crucial signaling step in more detail, the TrkA extracellular domain polypeptide (TrkA-RED) was overexpressed in Sf21 insect cells and purified to homogeneity. The recombinant TrkA-RED is a 70 kDa acidic glycoprotein with a pI of 5.1, and mimics the intact TrkA receptor for NGF binding with a dissociation constant, Kd, of 2.9 nM. Thus, the recombinant TrkA-RED is functionally competent and can be used to elucidate the interaction of NGF and TrkA receptor. Circular dichroism difference spectra indicated that, upon association of NGF with TrkA-RED, a minor conformational change occurred to form a complex with decreased ordered secondary structure. Interaction between NGF and TrkA-RED was also demonstrated by size exclusion chromatography, light scattering, and chemical crosslinking with evidence for formation of a higher molecular weight complex consistent with a (TrkA-RED)2-(NGF dimer) complex. Association and dissociation rates of 5.6 x 10(5) M(-1) s(-1) and 1.6 x 10(-3) s(-1), respectively, were determined by biosensor technology. Thus, initiation of signaling may stem from NGF-induced receptor dimerization concomitant with a small conformational change.

摘要

神经生长因子(NGF)是神经营养因子家族的原型,该家族支持重要的神经元程序,如交感神经、感觉神经和脑神经元亚群的分化与存活。NGF与两类细胞表面受体结合:p75LANR和p140TrkA。NGF与p140TrkA结合通过激活酪氨酸激酶活性启动神经元信号通路,随后通过磷酸化级联反应导致快速信号转导。为了更详细地研究这一关键信号步骤,TrkA细胞外结构域多肽(TrkA-RED)在Sf21昆虫细胞中过表达并纯化至同质。重组TrkA-RED是一种70 kDa的酸性糖蛋白,pI为5.1,模拟完整的TrkA受体与NGF结合,解离常数Kd为2.9 nM。因此,重组TrkA-RED在功能上具有活性,可用于阐明NGF与TrkA受体的相互作用。圆二色性差光谱表明,NGF与TrkA-RED结合时,发生了轻微的构象变化,形成了具有降低的有序二级结构的复合物。尺寸排阻色谱、光散射和化学交联也证明了NGF与TrkA-RED之间的相互作用,有证据表明形成了与(TrkA-RED)2-(NGF二聚体)复合物一致的更高分子量复合物。通过生物传感器技术测定的缔合和解离速率分别为5.6×10(5)M(-1)s(-1)和1.6×10(-3)s(-1)。因此,信号的启动可能源于NGF诱导的受体二聚化以及伴随的小构象变化。