Ivanov D E, Puchin'ian D M
Research Institute of Traumatology and Orthopedics, Saratov.
Usp Fiziol Nauk. 1998 Jan-Mar;29(1):58-71.
The study conducts the analysis of liver dysfunctions in traumatic stress. The severe craniocerebral injuries, the skeletal bone injuries and the prolonged crush syndrome cause dystrophic changes of hepatocytes. The biochemical data testify the cytolytic syndrome and the liver insufficiency in patients with the traumatic disease. The hepatocytes metabolism disturbances and morphologically expressed dystrophy prove the development of the "traumatic hepatosis".
该研究对创伤应激中的肝功能障碍进行了分析。严重颅脑损伤、骨骼损伤和长期挤压综合征会导致肝细胞营养不良性改变。生化数据证实了创伤性疾病患者存在细胞溶解综合征和肝功能不全。肝细胞代谢紊乱以及形态学上表现出的营养不良证明了“创伤性肝病”的发展。
