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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Nasu T, Sasaki M, Shibata H

Department of Veterinary Pharmacology, Faculty of Agriculture, Yamaguchi University, Japan.

Gen Pharmacol. 1998 Jul;31(1):43-5. doi: 10.1016/s0306-3623(97)00422-9.

Bay K 8644, a dihydropyridine Ca2+ agonist, (10(-6) M), induced strong rhythmic contractions in taenia coli and induced a contracture after about 5.5 hr of the application. Increases in Bay K 8644 concentrations prolonged the period of rhythmic contractions and slowed the onset of the contracture. 2. Nifedipine, a dihydropyridine Ca2+ antagonist, inhibited the delayed contracture to Bay K 8644 more than the initial rhythmic contractions. The contracture to Bay K 8644 may be preferentially dependent on extracellular Ca2+ entry in comparison with the rhythmic contractions in taenia coli.

Nasu T, Sasaki M, Shibata H

Department of Veterinary Pharmacology, Faculty of Agriculture, Yamaguchi University, Japan.

Gen Pharmacol. 1998 Jul;31(1):43-5. doi: 10.1016/s0306-3623(97)00422-9.

Bay K 8644, a dihydropyridine Ca2+ agonist, (10(-6) M), induced strong rhythmic contractions in taenia coli and induced a contracture after about 5.5 hr of the application. Increases in Bay K 8644 concentrations prolonged the period of rhythmic contractions and slowed the onset of the contracture. 2. Nifedipine, a dihydropyridine Ca2+ antagonist, inhibited the delayed contracture to Bay K 8644 more than the initial rhythmic contractions. The contracture to Bay K 8644 may be preferentially dependent on extracellular Ca2+ entry in comparison with the rhythmic contractions in taenia coli.