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臂丛神经产瘫继发的肩胛盂畸形

Glenoid deformity secondary to brachial plexus birth palsy.

作者信息

Pearl M L, Edgerton B W

机构信息

Department of Orthopaedics, Kaiser Permanente, Los Angeles Medical Center, California 90027, USA.

出版信息

J Bone Joint Surg Am. 1998 May;80(5):659-67. doi: 10.2106/00004623-199805000-00006.

DOI:10.2106/00004623-199805000-00006
PMID:9611026
Abstract

The association between internal rotation contracture secondary to brachial plexus birth palsy and deformity and posterior dislocation of the glenohumeral joint has been known for a long time. The precise nature of these deformities and their pathogenesis, however, remain unclear. Twenty-five children, ranging in age from 1.5 to 13.5 years, had an operation to release an internal rotation contracture secondary to brachial plexus birth palsy; eleven had a latissimus dorsi transfer to augment external rotation power as well. Arthrograms were made intraoperatively in order to clarify the pathological changes that occur in the glenohumeral joint during growth in patients who have this condition. Seven children had a concentric glenohumeral joint (the humeral head was well centered in the glenoid fossa). The remaining eighteen children (72 per cent) had a deformity of the posterior aspect of the glenoid. Five of these children had flattening of the posterior aspect of the glenoid, seven had a biconcave glenoid with the humeral head articulating with the posterior of the two concavities, and six had a so-called pseudoglenoid (the most severe deformity, in which the humeral head articulated with a distinct, retroverted, posterior articular surface). Internal rotation contracture secondary to brachial plexus birth palsy may lead to glenoid deformity that is severely advanced by the time that the child is two years old. In patients who have such a contracture, we recommend early imaging of the shoulder with arthrography or some other modality to allow visualization of the skeletally immature glenohumeral joint.

摘要

臂丛神经产瘫继发的内旋挛缩与盂肱关节畸形及后脱位之间的关联早已为人所知。然而,这些畸形的确切性质及其发病机制仍不清楚。25名年龄在1.5至13.5岁之间的儿童接受了手术,以松解臂丛神经产瘫继发的内旋挛缩;其中11名儿童还进行了背阔肌转移术以增强外旋力量。术中进行了关节造影,以明确患有这种疾病的患者在生长过程中盂肱关节发生的病理变化。7名儿童的盂肱关节呈同心圆状(肱骨头在关节盂窝内居中良好)。其余18名儿童(72%)存在关节盂后方畸形。其中5名儿童关节盂后方扁平,7名儿童关节盂呈双凹形,肱骨头与两个凹面中的后方凹面相关节,6名儿童存在所谓的假关节盂(最严重的畸形,其中肱骨头与一个明显的、后倾的后方关节面相关节)。臂丛神经产瘫继发的内旋挛缩可能导致关节盂畸形,到患儿两岁时这种畸形已严重进展。对于患有这种挛缩的患者,我们建议早期用关节造影或其他方式对肩部进行成像,以便观察骨骼未成熟的盂肱关节。

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