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急性铊中毒的初始感觉运动和迟发性自主神经病变

Initial sensorimotor and delayed autonomic neuropathy in acute thallium poisoning.

作者信息

Nordentoft T, Andersen E B, Mogensen P H

机构信息

Department of Nephrology, Hvidovre University Hospital, Copenhagen, Denmark.

出版信息

Neurotoxicology. 1998 Jun;19(3):421-6.

PMID:9621348
Abstract

In a 27-year old male with acute thallium poisoning, signs of initially severe sensorimotor neuropathy with complete remission after two weeks were demonstrated. Signs of cardiovascular autonomic neuropathy were initially absent, but developed after a latency period of one week with marked improvement after seven months. Delayed autonomic neuropathy may be caused by a late affection of small unmyelinated autonomic nerve fibers.

摘要

在一名27岁的急性铊中毒男性患者中,起初表现为严重的感觉运动神经病变,两周后完全缓解。心血管自主神经病变起初并不明显,但在一周的潜伏期后出现,七个月后有显著改善。迟发性自主神经病变可能是由小型无髓鞘自主神经纤维的晚期损伤所致。

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Initial sensorimotor and delayed autonomic neuropathy in acute thallium poisoning.急性铊中毒的初始感觉运动和迟发性自主神经病变
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