Chloride channels and endocytosis: ClC-5 makes a dent.
作者信息
George A L
机构信息
Departments of Medicine and Pharmacology, Vanderbilt University Medical Center, Nashville, TN 37232, USA.
出版信息
Proc Natl Acad Sci U S A. 1998 Jul 7;95(14):7843-5. doi: 10.1073/pnas.95.14.7843.
Abstract
摘要
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本文引用的文献
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ClC-5, the chloride channel mutated in Dent's disease, colocalizes with the proton pump in endocytotically active kidney cells.氯离子通道蛋白5（ClC-5）在丹特病（Dent's disease）中发生突变，它在具有活跃内吞作用的肾细胞中与质子泵共定位。
Proc Natl Acad Sci U S A. 1998 Jul 7;95(14):8075-80. doi: 10.1073/pnas.95.14.8075.
2
Evidence for the intracellular location of chloride channel (ClC)-type proteins: co-localization of ClC-6a and ClC-6c with the sarco/endoplasmic-reticulum Ca2+ pump SERCA2b.氯离子通道（ClC）型蛋白细胞内定位的证据：ClC-6a和ClC-6c与肌浆网/内质网Ca2+泵SERCA2b的共定位。
Biochem J. 1998 Mar 1;330 ( Pt 2)(Pt 2):1015-21. doi: 10.1042/bj3301015.
3
X-linked hypercalciuric nephrolithiasis: clinical syndromes and chloride channel mutations.X连锁高钙尿性肾结石病：临床综合征与氯离子通道突变
Kidney Int. 1998 Jan;53(1):3-17. doi: 10.1046/j.1523-1755.1998.00718.x.
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Pore-forming segments in voltage-gated chloride channels.电压门控氯离子通道中的成孔片段。
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