Wilson J R, Stittsworth J D, Kadir A, Fisher M A
Department of Neurology, Hines VA Hospital, Illinois 60141, USA.
Muscle Nerve. 1998 Sep;21(9):1228-30. doi: 10.1002/(sici)1097-4598(199809)21:9<1228::aid-mus20>3.0.co;2-m.
Motor conduction velocities (CVs) were correlated with distal compound muscle action potential (CMAP) amplitudes for tibial, peroneal, and median nerves in patients with biopsy-proven chronic inflammatory demyelinating polyneuropathy (CIDP), diabetic neuropathy, and amyotrophic lateral sclerosis. Only in the diabetic patients did CV significantly correlate with CMAP amplitude. The data show that diabetic neuropathy produces conduction velocity slowing that cannot be explained by axon loss alone, and that differentiation between diabetic neuropathy and CIDP in an individual nerve is difficult.
对经活检证实患有慢性炎症性脱髓鞘性多发性神经病（CIDP）、糖尿病性神经病和肌萎缩侧索硬化症的患者，检测其胫神经、腓总神经和正中神经的运动传导速度（CVs），并将其与远端复合肌肉动作电位（CMAP）波幅进行相关性分析。仅在糖尿病患者中，CV与CMAP波幅显著相关。数据表明，糖尿病性神经病导致的传导速度减慢不能仅用轴突丢失来解释，而且在单根神经中区分糖尿病性神经病和CIDP很困难。
