[Flowmeter changes induced by glucagon in hepato-portal hemodynamics in the course of hemorrhagic shock in dogs].

Anoxic lesion in haemorrhagic shock may result in exclusion of the capillary circulation to the point of stagnant hypoxia. Under normal conditions, glucagon regulates hepatoportal haemodynamics. Its experimentation in the correction of haemorrhagic shock in the dog showed that it increased hepatoportal flow towards normality by reducing local resistance. The consequences with respect to intestinal tissue metabolism make this finding of particular interest.