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Dysfunctional factor VII variant (FVII Tondabayashi) with R79Q: determination of mutated site with monoclonal anti-human factor VII antibody (B101/B1).

作者信息

Takamiya O, Takeuchi S

机构信息

Department of Medical Technology, School of Allied Medical Sciences, Shinshu University, Matsumoto, Japan.

出版信息

Clin Chem. 1998 Sep;44(9):1993-5.

PMID:9732992
Abstract
摘要

相似文献

1
Dysfunctional factor VII variant (FVII Tondabayashi) with R79Q: determination of mutated site with monoclonal anti-human factor VII antibody (B101/B1).具有R79Q的功能失调性因子VII变异体(FVII 丰田):用单克隆抗人因子VII抗体(B101/B1)确定突变位点
Clin Chem. 1998 Sep;44(9):1993-5.
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Surface plasmon resonance studies of the interaction between factor VII and tissue factor. Demonstration of defective tissue factor binding in a variant FVII molecule (FVII-R79Q).凝血因子VII与组织因子相互作用的表面等离子体共振研究。变异型FVII分子(FVII-R79Q)中组织因子结合缺陷的证明。
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引用本文的文献

1
Conformational Changes of Congenital FVII Variants with Defective Binding to Tissue Factor ARG304GLN (FVII Padua), ARG 304TRP (FVII Nagoya) and ARG79GLN (FVII Shinjo or Tondabayashi).与组织因子结合缺陷的先天性FVII变体(ARG304GLN(FVII帕多瓦)、ARG 304TRP(FVII名古屋)和ARG79GLN(FVII新庄或富田林))的构象变化
Int J Biomed Sci. 2013 Dec;9(4):185-93.