Gisiger V
Département de Pathologie et Biologie cellulaire, Université de Montréal, Québec, Canada.
J Physiol Paris. 1998 Jun-Aug;92(3-4):163-6. doi: 10.1016/s0928-4257(98)80003-x.
Earlier studies in the sympathetic ganglion have led to the proposal that adaptation of transcription to trans-synaptic activity is controlled by a signal transduction pathway featuring a transcription factor which translocates to the nucleus upon its release from the post-synaptic membrane by after-hyperpolarization. In light of recent progress, it is proposed here that NF-kappa B constitutes the postulated transcription factor.